Normal human immunoglobulin G4 is bispecific: it has two different antigen-combining sites. by Schuurman, J, Van Ree, R, Perdok, G, Van Doorn, H, Tan, K, Aalberse, R

Phenotypic differences between IgG4+ and IgG1+ B cells point to distinct regulation of the IgG4 response. by Lighaam, L, Vermeulen, E, Bleker, T, Meijlink, K, Aalberse, R, Barnes, E, Culver, E, van Ham, S, Rispens, T

Increases in IgE, Eosinophils, and Mast Cells can be Used in Diagnosis and to Predict Relapse of IgG4-related Disease. by Culver, E, Sadler, R, Bateman, A, Makuch, M, Cargill, T, Ferry, B, Aalberse, R, Barnes, E, Rispens, T

Phenotypic differences between IgG4 + and IgG 1 + B cells point to distinct regulation of the IgG 4 response by Lighaam, L, Vermeulen, E, Bleker, T, Meijlink, K, Aalberse, R, Barnes, E, Culver, E, Van Ham, S, Rispens, T

Altered FC and FAB glycosylation status in patients with IGG4-related sclerosing cholangitis and autoimmune pancreatitis by Culver, E, Van De Bovenkamp, F, Derksen, N, Cargill, T, Koeleman, C, Neef, L, Barnes, E, Aalberse, R, Wuhrer, M, Rispens, T

Increased IgG4 responses to multiple food and animal antigens indicate a polyclonal expansion and differentiation of pre-existing B cells in IgG4-related disease by Culver, E, Vermeulen, E, Makuch, M, van Leeuwen, A, Sadler, R, Cargill, T, Klenerman, P, Aalberse, R, van Ham, S, Barnes, E, Rispens, T

Unique patterns of glycosylation in immunoglobulin subclass G4‐related disease and primary sclerosing cholangitis by Culver, E, Van De Bovenkamp, F, Derksen, N, Koers, J, Cargill, T, Barnes, E, De Neef, L, Koeleman, C, Aalberse, R, Wuhrer, M, Rispens, T