Albert Erives

Albert Erives (born March 4, 1972) is a developmental geneticist who studies transcriptional enhancers underlying animal development and diseases of development (cancers). Erives also proposed the pacRNA model for the dual origin of the genetic code and universal homochirality. He is known for work at the intersection of genetics, evolution, developmental biology, and gene regulation. He has worked at the California Institute of Technology, University of California, Berkeley, and Dartmouth College, and is an associate professor at the University of Iowa.

Erives has shown how genes of the nucleocytoplasmic large DNA viruses inform on intermediate steps in the evolution of the linear, chromatinized eukaryotic chromosome and its mechanisms of gene regulation. Provided by Wikipedia
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  1. 1
    A Screen for Gene Paralogies Delineating Evolutionary Branching Order of Early Metazoa

    A Screen for Gene Paralogies Delineating Evolutionary Branching Order of Early Metazoa by Albert Erives, Bernd Fritzsch

    Published 2020-02-01
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  2. 2
    Episodic evolution of a eukaryotic NADK repertoire of ancient provenance.

    Episodic evolution of a eukaryotic NADK repertoire of ancient provenance. by Oliver Vickman, Albert Erives

    Published 2019-01-01
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  3. 3
    A closer look at the eve stripe 2 enhancers of Drosophila and Themira.

    A closer look at the eve stripe 2 enhancers of Drosophila and Themira. by Justin Crocker, Albert Erives

    Published 2008-11-01
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  4. 4
    Evolution acts on enhancer organization to fine-tune gradient threshold readouts.

    Evolution acts on enhancer organization to fine-tune gradient threshold readouts. by Justin Crocker, Yoichiro Tamori, Albert Erives

    Published 2008-11-01
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  5. 5
    Microsatellite repeat instability fuels evolution of embryonic enhancers in Hawaiian Drosophila.

    Microsatellite repeat instability fuels evolution of embryonic enhancers in Hawaiian Drosophila. by Andrew Brittain, Elizabeth Stroebele, Albert Erives

    Published 2014-01-01
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  6. 6
    Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO).

    Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO). by Allison J Cox, Benjamin W Darbro, Ronald M Laxer, Gabriel Velez, Xinyu Bing, Alexis L Finer, Albert Erives, Vinit B Mahajan, Alexander G Bassuk, Polly J Ferguson

    Published 2017-01-01
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  7. 7
    Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO).

    Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO). by Allison J Cox, Benjamin W Darbro, Ronald M Laxer, Gabriel Velez, Xinyu Bing, Alexis L Finer, Albert Erives, Vinit B Mahajan, Alexander G Bassuk, Polly J Ferguson

    Published 2017-01-01
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