Streptozotocin-induced diabetes mellitus increases the risk for atrial fibrillation by Ziberna, K, Recalde, A, Carnicer, R, Casadei, B

BH4 supplementation as a new treatment for diabetic cardiomyopathy by Carnicer, R, Ziberna, K, Arya, R, Channon, K, Casadei, B

Nitric oxide synthases in heart failure. by Carnicer, R, Crabtree, M, Sivakumaran, V, Casadei, B, Kass, D

Is depressed myocyte contractility an early event in the natural history of heart failure? by Idigo, W, Zhang, Y, Lygate, C, Carnicer, R, Dawson, D, Casadei, B

Is depressed myocyte contractility an early event in the natural history of heart failure? by Idigo, W, Zhang, Y, Lygate, C, Carnicer, R, Dawson, D, Casadei, B

P683The role of nitric oxide synthase (NOS) and its essential cofactor tetrahydrobiopterin (BH4) in diabetic cardiomyopathy. by Duglan, D, Carnicer, R, Simon, J, Chuaiphichai, S, Hale, A, Channon, K, Casadei, B

Cardiomyocyte-Targeted Overexpression of GTP Cyclohydrolase-1 Increases Myocyte Lusitropy and Reduces Superoxide Production by Carnicer, R, Hale, AB, Liu, X, Lim, G, Alp, N, Channon, K, Casadei, B

200 Nitric oxide promotes insulin-independent glucose uptake and preserves cardiac function and energetics in diabetes by Ziberna, K, Duglan, D, Mafrici, S, Simon, J, Lygate, C, Channon, K, Casadei, B, Carnicer, R

Evaluation of the role of miR-31-dependent reduction in dystrophin and nNOS on atrial-fibrillation-induced electrical remodelling in man by Reilly, S, Liu, X, Carnicer, R, Rajakumar, T, Sayeed, R, Krasopoulos, G, Verheule, S, Fulga, T, Schotten, U, Casadei, B

Cardiomyocyte-targeted overexpression of GTP cyclohydrolase-1 increases nNOS activity and hastens myocardial relaxation by Carnicer, R, Hale, AB, Liu, X, Bendall, J, Lim, G, Alp, N, Channon, K, Casadei, B

Fast, quantitative, murine cardiac 19F MRI/MRS of PFCE-labeled progenitor stem cells and macrophages at 9.4T by Constantinides, C, Maguire, M, McNeill, E, Carnicer, R, Swider, E, Srinivas, M, Carr, C, Schneider, J

Regulation of endothelial nitric-oxide synthase (NOS) S-glutathionylation by neuronal NOS: evidence of a functional interaction between myocardial constitutive NOS isoforms. by Idigo, W, Reilly, S, Zhang, M, Zhang, Y, Jayaram, R, Carnicer, R, Crabtree, M, Balligand, J, Casadei, B

In vivo tracking and 1H/19F magnetic resonance imaging of biodegradable polyhydroxyalkanoate/polycaprolactone blend scaffolds seeded with labeled cardiac stem cells by Constantinides, C, Basnett, P, Lukasiewicz, B, Carnicer, R, Swider, E, Majid, Q, Srinivas, M, Carr, C, Roy, I

The subcellular localisation of neuronal nitric oxide synthase determines the downstream effects of NO on myocardial function by Carnicer, R, Suffredini, S, Liu, X, Reilly, S, Simon, J, Surdo, N, Zhang, Y, Lygate, C, Channon, K, Casadei, B

Endothelial cell-specific roles for tetrahydrobiopterin in myocardial function, cardiac hypertrophy, and response to myocardial ischemia-reperfusion injury by Chuaiphichai, S, Chu, SM, Carnicer, R, Kelly, M, Bendall, JK, Simon, JN, Douglas, G, Crabtree, MJ, Casadei, B, Channon, KM

NO from neuronal NO synthase increases after beta-adrenergic stimulation but does not control mitochondrial respiration by Bergem, S, Kohlhaas, M, Nickel, A, Meiser, M, Hohl, M, Carnicer, R, Laufs, U, Boehm, M, Casadei, B, Maack, C

Inducibility, but not stability, of atrial fibrillation is increased by NOX2 overexpression in mice by Mighiu, AS, Recalde, A, Ziberna, K, Carnicer, R, Tomek, J, Bub, G, Brewer, AC, Verheule, S, Shah, AM, Simon, JN, Casadei, B

Human ischemic cardiomyopathy shows cardiac nos1 translocation and its increased levels are related to left ventricular performance by Roselló-Lletí, E, Carnicer, R, Tarazón, E, Ortega, A, Gil-Cayuela, C, Lago, F, González-Juanatey, J, Portolés, M, Rivera, M

Tetrahydrobiopterin protects against hypertrophic heart disease independent of myocardial nitric oxide synthase coupling. by Hashimoto, T, Sivakumaran, V, Carnicer, R, Zhu, G, Hahn, V, Bedja, D, Recalde, A, Duglan, D, Channon, K, Casadei, B, Kass, D

Tetrahydrobiopterin protects against hypertrophic heart disease independent of myocardial nitric oxide synthase coupling by Hashimoto, T, Sivakumaran, V, Carnicer, R, Zhu, G, Hahn, V, Bedja, D, Recalde, A, Duglan, D, Channon, K, Casadei, B, Kass, D