Acidosis differentially modulates inactivation in NaV1.2, NaV1.4, and NaV1.5 channels by Yury Y. Vilin, Colin H. Peters, Peter eRuben

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Depolarization of the conductance-voltage relationship in the NaV1.5 mutant, E1784K, is due to altered fast inactivation. by Colin H Peters, Alec Yu, Wandi Zhu, Jonathan R Silva, Peter C Ruben

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Regulation of HCN Channels by Protein Interactions by Colin H. Peters, Rohit K. Singh, John R. Bankston, Catherine Proenza, Catherine Proenza

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Intracellular Na⁺ Modulates Pacemaking Activity in Murine Sinoatrial Node Myocytes: An <i>In Silico</i> Analysis by Stefano Morotti, Haibo Ni, Colin H. Peters, Christian Rickert, Ameneh Asgari-Targhi, Daisuke Sato, Alexey V. Glukhov, Catherine Proenza, Eleonora Grandi

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LRMP inhibits cAMP potentiation of HCN4 channels by disrupting intramolecular signal transduction by Colin H Peters, Rohit K Singh, Avery A Langley, William G Nichols, Hannah R Ferris, Danielle A Jeffrey, Catherine Proenza, John R Bankston

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The L1624Q Variant in SCN1A Causes Familial Epilepsy Through a Mixed Gain and Loss of Channel Function by Laura B. Jones, Colin H. Peters, Richard E. Rosch, Richard E. Rosch, Maxine Owers, Elaine Hughes, Elaine Hughes, Deb K. Pal, Deb K. Pal, Peter C. Ruben

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