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  • Dawn Sijin Nin
Showing 1 - 5 results of 5 for search 'Dawn Sijin Nin', query time: 0.03s Refine Results
  1. 1
    Biology of Cancer-Testis Antigens and Their Therapeutic Implications in Cancer

    Biology of Cancer-Testis Antigens and Their Therapeutic Implications in Cancer by Dawn Sijin Nin, Lih-Wen Deng

    Published 2023-03-01
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    Article
  2. 2
    Misfolded N-CoR is linked to the ectopic reactivation of CD34/Flt3-based stem-cell phenotype in promyelocytic and monocytic acute myeloid leukemia

    Misfolded N-CoR is linked to the ectopic reactivation of CD34/Flt3-based stem-cell phenotype in promyelocytic and monocytic acute myeloid leukemia by Dawn Sijin Nin, Li eFeng, Sridevi eVisvanathan, Matiullah eKhan

    Published 2015-10-01
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    Article
  3. 3
    Role of misfolded N-CoR mediated transcriptional deregulation of Flt3 in acute monocytic leukemia (AML)-M5 subtype.

    Role of misfolded N-CoR mediated transcriptional deregulation of Flt3 in acute monocytic leukemia (AML)-M5 subtype. by Dawn Sijin Nin, Wai Kay Kok, Feng Li, Shinichiro Takahashi, Wee Joo Chng, Matiullah Khan

    Published 2012-01-01
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    Article
  4. 4
    BRD4 Phosphorylation Regulates HPV E2-Mediated Viral Transcription, Origin Replication, and Cellular MMP-9 Expression

    BRD4 Phosphorylation Regulates HPV E2-Mediated Viral Transcription, Origin Replication, and Cellular MMP-9 Expression by Shwu-Yuan Wu, Dawn Sijin Nin, A-Young Lee, Scott Simanski, Thomas Kodadek, Cheng-Ming Chiang

    Published 2016-08-01
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    Article
  5. 5
    Akt-induced phosphorylation of N-CoR at serine 1450 contributes to its misfolded conformational dependent loss (MCDL) in acute myeloid leukemia of the M5 subtype.

    Akt-induced phosphorylation of N-CoR at serine 1450 contributes to its misfolded conformational dependent loss (MCDL) in acute myeloid leukemia of the M5 subtype. by Dawn Sijin Nin, Azhar Bin Ali, Koichi Okumura, Norio Asou, Chien-Shing Chen, Wee Joo Chng, Matiullah Khan

    Published 2013-01-01
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    Article

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