Extracellular calreticulin is present in the joints of patients with rheumatoid arthritis and inhibits FasL (CD95L)-mediated apoptosis of T cells. by Tarr, J, Winyard, P, Ryan, B, Harries, L, Haigh, R, Viner, N, Eggleton, P

Oxidative and Nitrative Modification of C1q Affects its Structure, Function and Antigenicity in Systemic Lupus Erythematosus by Ryan, B, Eggleton, P, Brown, S, Viner, N, Nissim, A, Isenberg, D, Haigh, R, Winyard, P

POST-TRANSLATIONAL MODIFICATIONS OF C1Q LEAD TO ANTIGENICITY AND BREAKDOWN OF IMMUNE TOLERANCE IN SYSTEMIC LUPUS ERYTHEMATOSUS by Eggleton, P, Ryan, B, Brown, S, Johnson, S, Viner, N, Nissim, A, Isenberg, D, Haigh, R, Winyard, P

A mechanism of release of calreticulin from cells during apoptosis. by Tarr, J, Young, P, Morse, R, Shaw, D, Haigh, R, Petrov, P, Johnson, S, Winyard, P, Eggleton, P

Changes in apoptotic gene expression in lymphocytes from rheumatoid arthritis and systemic lupus erythematosus patients compared with healthy lymphocytes. by Eggleton, P, Harries, L, Alberigo, G, Wordsworth, P, Viner, N, Haigh, R, Donnelly, S, Jones, H, Chikanza, I, O'Conner, T, Thomson, A, Winyard, P

Measurement and meaning of markers of reactive species of oxygen, nitrogen and sulfur in healthy human subjects and patients with inflammatory joint disease. by Winyard, P, Ryan, B, Eggleton, P, Nissim, A, Taylor, E, Lo Faro, M, Burkholz, T, Szabó-Taylor, K, Fox, B, Viner, N, Haigh, R, Benjamin, N, Jones, A, Whiteman, M