Comparison of different therapies for neonatal diabetes – glibenclamide vs insulin by Iberl, M

Mice expressing a human K(ATP) channel mutation have altered channel ATP sensitivity but no cardiac abnormalities. by Clark, R, Männikkö, R, Stuckey, D, Iberl, M, Clarke, K, Ashcroft, F

FTO is expressed in neurones throughout the brain and its expression is unaltered by fasting. by McTaggart, J, Lee, S, Iberl, M, Church, C, Cox, R, Ashcroft, F

Mice expressing a human K ATP channel mutation have altered channel ATP sensitivity but no cardiac abnormalities by Clark, R, Männikkö, R, Stuckey, D, Iberl, M, Clarke, K, Ashcroft, F

Muscle dysfunction caused by a KATP channel mutation in neonatal diabetes is neuronal in origin. by Clark, R, McTaggart, J, Webster, R, Mannikko, R, Iberl, M, Sim, X, Rorsman, P, Glitsch, M, Beeson, D, Ashcroft, F

A mouse model of human hyperinsulinism produced by the E1506K mutation in the sulphonylurea receptor SUR1. by Shimomura, K, Tusa, M, Iberl, M, Brereton, M, Kaizik, S, Proks, P, Lahmann, C, Yaluri, N, Modi, S, Huopio, H, Ustinov, J, Otonkoski, T, Laakso, M, Ashcroft, F

Hyperglycaemia induces metabolic dysfunction and glycogen accumulation in pancreatic β-cells by Brereton, M, Rohm, M, Shimomura, K, Holland, C, Tornovsky-Babeay, S, Dadon, D, Iberl, M, Chibalina, M, Lee, S, Glaser, B, Dor, Y, Rorsman, P, Clark, A, Ashcroft, F

Reversible changes in pancreatic islet structure and function produced by elevated blood glucose by Brereton, M, Iberl, M, Shimomura, K, Zhang, Q, Adriaenssens, A, Proks, P, Spiliotis, I, Dace, W, Mattis, K, Ramracheya, R, Gribble, F, Reimann, F, Clark, A, Rorsman, P, Ashcroft, F

Reversible changes in pancreatic islet structure and function produced by elevated blood glucose. by Brereton, M, Iberl, M, Shimomura, K, Zhang, Q, Adriaenssens, A, Proks, P, Spiliotis, I, Dace, W, Mattis, K, Ramracheya, R, Gribble, F, Reimann, F, Clark, A, Rorsman, P, Ashcroft, F