Prikaz rezultata 1 – 20 od 37 za pretragu 'Jonathan D Cooper', vrijeme upita: 0,06s Detaljiziraj rezultate
  1. 1
    Glial Dysfunction and Its Contribution to the Pathogenesis of the Neuronal Ceroid Lipofuscinoses

    Glial Dysfunction and Its Contribution to the Pathogenesis of the Neuronal Ceroid Lipofuscinoses od Keigo Takahashi, Hemanth R. Nelvagal, Jenny Lange, Jonathan D. Cooper, Jonathan D. Cooper, Jonathan D. Cooper

    Izdano 2022-04-01
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  2. 2
    Top-down and bottom-up propagation of disease in the neuronal ceroid lipofuscinoses

    Top-down and bottom-up propagation of disease in the neuronal ceroid lipofuscinoses od John R. Ostergaard, Hemanth R. Nelvagal, Hemanth R. Nelvagal, Jonathan D. Cooper, Jonathan D. Cooper, Jonathan D. Cooper

    Izdano 2022-11-01
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  3. 3
    Combined Anti-inflammatory and Neuroprotective Treatments Have the Potential to Impact Disease Phenotypes in Cln3−/− Mice

    Combined Anti-inflammatory and Neuroprotective Treatments Have the Potential to Impact Disease Phenotypes in Cln3−/− Mice od Marta A. Tarczyluk-Wells, Christoph Salzlechner, Allison R. Najafi, Ming J. Lim, Ming J. Lim, David Smith, Frances M. Platt, Brenda P. Williams, Jonathan D. Cooper, Jonathan D. Cooper, Jonathan D. Cooper

    Izdano 2019-09-01
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  4. 4
    Regional and cellular neuropathology in the palmitoyl protein thioesterase-1 null mutant mouse model of infantile neuronal ceroid lipofuscinosis

    Regional and cellular neuropathology in the palmitoyl protein thioesterase-1 null mutant mouse model of infantile neuronal ceroid lipofuscinosis od Ellen Bible, Praveena Gupta, Sandra L Hofmann, Jonathan D Cooper

    Izdano 2004-07-01
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  5. 5
    Anti-inflammatory impact of minocycline in a mouse model of tauopathy

    Anti-inflammatory impact of minocycline in a mouse model of tauopathy od Claire J Garwood, Jonathan D Cooper, Diane P Hanger, Wendy Noble

    Izdano 2010-10-01
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  6. 6
    Loss of amyloid precursor protein in a mouse model of Niemann–Pick type C disease exacerbates its phenotype and disrupts tau homeostasis

    Loss of amyloid precursor protein in a mouse model of Niemann–Pick type C disease exacerbates its phenotype and disrupts tau homeostasis od Ana Nunes, Sarah N.R. Pressey, Jonathan D. Cooper, Salvador Soriano

    Izdano 2011-06-01
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  7. 7
    Selective spatiotemporal patterns of glial activation and neuron loss in the sensory thalamocortical pathways of neuronal ceroid lipofuscinosis 8 mice

    Selective spatiotemporal patterns of glial activation and neuron loss in the sensory thalamocortical pathways of neuronal ceroid lipofuscinosis 8 mice od Mervi Kuronen, Anna-Elina Lehesjoki, Anu Jalanko, Jonathan D. Cooper, Outi Kopra

    Izdano 2012-09-01
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  8. 8
    Thalamocortical neuron loss and localized astrocytosis in the Cln3Δex7/8 knock-in mouse model of Batten disease

    Thalamocortical neuron loss and localized astrocytosis in the Cln3Δex7/8 knock-in mouse model of Batten disease od Charlie C. Pontikis, Susan L. Cotman, Marcy E. MacDonald, Jonathan D. Cooper

    Izdano 2005-12-01
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  9. 9
    Neurodegeneration and Epilepsy in a Zebrafish Model of CLN3 Disease (Batten Disease).

    Neurodegeneration and Epilepsy in a Zebrafish Model of CLN3 Disease (Batten Disease). od Kim Wager, Anselm A Zdebik, Sonia Fu, Jonathan D Cooper, Robert J Harvey, Claire Russell

    Izdano 2016-01-01
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  10. 10
    Early glial activation, synaptic changes and axonal pathology in the thalamocortical system of Niemann–Pick type C1 mice

    Early glial activation, synaptic changes and axonal pathology in the thalamocortical system of Niemann–Pick type C1 mice od Sarah N.R. Pressey, David A. Smith, Andrew M.S. Wong, Frances M. Platt, Jonathan D. Cooper

    Izdano 2012-03-01
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  11. 11
    Cellular trafficking and fate mapping of cells within the nervous system after in utero hematopoietic cell transplantation

    Cellular trafficking and fate mapping of cells within the nervous system after in utero hematopoietic cell transplantation od Matthew T. Grant, Hemanth Ramesh Nelvagal, Maria Tecos, Amal Hamed, Kerry Swanson, Jonathan D. Cooper, Jesse D. Vrecenak

    Izdano 2024-12-01
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  12. 12
    A murine model of variant late infantile ceroid lipofuscinosis recapitulates behavioral and pathological phenotypes of human disease.

    A murine model of variant late infantile ceroid lipofuscinosis recapitulates behavioral and pathological phenotypes of human disease. od Jeremy P Morgan, Helen Magee, Andrew Wong, Tarah Nelson, Bettina Koch, Jonathan D Cooper, Jill M Weimer

    Izdano 2013-01-01
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  13. 13
    Disruption of the autophagy-lysosome pathway is involved in neuropathology of the nclf mouse model of neuronal ceroid lipofuscinosis.

    Disruption of the autophagy-lysosome pathway is involved in neuropathology of the nclf mouse model of neuronal ceroid lipofuscinosis. od Melanie Thelen, Markus Damme, Michaela Schweizer, Christian Hagel, Andrew M S Wong, Jonathan D Cooper, Thomas Braulke, Giovanna Galliciotti

    Izdano 2012-01-01
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  14. 14
    Glial activation spreads from specific cerebral foci and precedes neurodegeneration in presymptomatic ovine neuronal ceroid lipofuscinosis (CLN6)

    Glial activation spreads from specific cerebral foci and precedes neurodegeneration in presymptomatic ovine neuronal ceroid lipofuscinosis (CLN6) od Manfred J. Oswald, David N. Palmer, Graham W. Kay, Stephen J.A. Shemilt, Payam Rezaie, Jonathan D. Cooper

    Izdano 2005-10-01
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  15. 15
    Compromised astrocyte function and survival negatively impact neurons in infantile neuronal ceroid lipofuscinosis

    Compromised astrocyte function and survival negatively impact neurons in infantile neuronal ceroid lipofuscinosis od Jenny Lange, Luke J. Haslett, Emyr Lloyd-Evans, Jennifer M. Pocock, Mark S. Sands, Brenda P. Williams, Jonathan D. Cooper

    Izdano 2018-08-01
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  16. 16
    Genetically Corrected iPSC-Derived Neural Stem Cell Grafts Deliver Enzyme Replacement to Affect CNS Disease in Sanfilippo B Mice

    Genetically Corrected iPSC-Derived Neural Stem Cell Grafts Deliver Enzyme Replacement to Affect CNS Disease in Sanfilippo B Mice od Don Clarke, Yewande Pearse, Shih-hsin Kan, Steven Q. Le, Valentina Sanghez, Jonathan D. Cooper, Patricia I. Dickson, Michelina Iacovino

    Izdano 2018-09-01
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  17. 17
    Exacerbated neuronal ceroid lipofuscinosis phenotype in Cln1/5 double-knockout mice

    Exacerbated neuronal ceroid lipofuscinosis phenotype in Cln1/5 double-knockout mice od Tea Blom, Mia-Lisa Schmiedt, Andrew M. Wong, Aija Kyttälä, Jarkko Soronen, Matti Jauhiainen, Jaana Tyynelä, Jonathan D. Cooper, Anu Jalanko

    Izdano 2013-03-01
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  18. 18
    Progressive thalamocortical neuron loss in Cln5 deficient mice: Distinct effects in Finnish variant late infantile NCL

    Progressive thalamocortical neuron loss in Cln5 deficient mice: Distinct effects in Finnish variant late infantile NCL od Carina von Schantz, Catherine Kielar, Stine N. Hansen, Charlie C. Pontikis, Noreen A. Alexander, Outi Kopra, Anu Jalanko, Jonathan D. Cooper

    Izdano 2009-05-01
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  19. 19
    Effects of chronic cannabidiol in a mouse model of naturally occurring neuroinflammation, neurodegeneration, and spontaneous seizures

    Effects of chronic cannabidiol in a mouse model of naturally occurring neuroinflammation, neurodegeneration, and spontaneous seizures od Joshua T. Dearborn, Hemanth R. Nelvagal, Nicholas R. Rensing, Keigo Takahashi, Stephanie M. Hughes, Thomas M. Wishart, Jonathan D. Cooper, Michael Wong, Mark S. Sands

    Izdano 2022-07-01
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  20. 20
    Visual deficits in a mouse model of Batten disease are the result of optic nerve degeneration and loss of dorsal lateral geniculate thalamic neurons

    Visual deficits in a mouse model of Batten disease are the result of optic nerve degeneration and loss of dorsal lateral geniculate thalamic neurons od Jill M. Weimer, Andrew W. Custer, Jared W. Benedict, Noreen A. Alexander, Evan Kingsley, Howard J. Federoff, Jonathan D. Cooper, David A. Pearce

    Izdano 2006-05-01
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