THE ALZHEIMER’S DISEASE MULTIFACTORIAL THERAPEUTIC AGENT E2F4DN PREVENTS THE UPREGULATION OF THE PRO-DEATH GENE CITED 2 IN RESPONSE TO DNA DAMAGE IN NEURONS by Aina Maria Llabrés Mas, Alberto Garrido García, Vanesa Cano Daganzo, Jose María Frade

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E2F4-BASED GENE THERAPY MITIGATES THE IMMUNE RESPONSE OF THE ALZHEIMER’S DISEASE MOUSE MODEL 5XFAD by Morgan Ramón Landreau, Noelia López-Sánchez, Alberto Garrido García, Vanesa Cano Daganzo, Jose María Frade

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E2F4DN-BASED GENE THERAPY PREVENTS LONG-TERM POTENTIATION AND COGNITIVE LOSS AND IMPROVES NEURONAL SURVIVAL IN HOMOZYGOUS 5XFAD MICE. by Cristina Sánchez-Puelles, Alberto Fraj, Vanesa Cano Daganzo, Alberto Garrido García, Gertrudis Perea, Jose María Frade

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NEURONAL EXPRESSION OF A MUTANT FORM OF E2F4 RESTORES ADULT NEUROGENESIS IN A MOUSE MODEL OF ALZHEIMER’S DISEASE by Anna Lozano Ureña, Aina Maria Llabrés Mas, Morgan Ramón-Landreau, Cristina Sánchez-Puelles, Vanesa Cano Daganzo, Jose María Frade

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Neuronal injury external to the retina rapidly activates retinal glia, followed by elevation of markers for cell cycle re-entry and death in retinal ganglion cells. by Alba Galan, Pauline Dergham, Pedro Escoll, Antonio de-la-Hera, Philippe M D'Onofrio, Mark M Magharious, Paulo D Koeberle, José María Frade, H Uri Saragovi

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