Where have all the stem cells gone? by Bagi, Z, Kaley, G

Overexpression of Arginase I Leads to Diminished Acetylcholine-Induced Coronary Arteriolar Dilation in Patients with Type 2 Diabetes Mellitus by Bagi, Z, Beleznai, T, Kaley, G

Type 2 diabetic mice have increased blood pressure and enhanced arteriolar basal tone due to COX-2-dependent TxA(2)/PGH(2) production by Bagi, Z, Koller, A, Kaley, G

Specific roles of COX-1 and COX-2 inhibitors to interfere with the regulation of microvascular resistance in normal mice and mice with type 2 diabetes mellitus by Bagi, Z, Koller, A, Kaley, G

Superoxide-NO interaction decreases flow- and agonist-induced dilations of coronary arterioles in Type 2 diabetes mellitus. by Bagi, Z, Koller, A, Kaley, G

PPARgamma activation, by reducing oxidative stress, increases NO bioavailability in coronary arterioles of mice with Type 2 diabetes. by Bagi, Z, Koller, A, Kaley, G

PPAR gamma activation improves wall shear stress dependent regulation of coronary arteriolar diameter in type 2 diabetes mellitus by Bagi, Z, Koller, A, Kaley, G

PPAR gamma activation restores NO-Mediated dilations of coronary Arterioles in type 2 diabetes Mellitus by Bagi, Z, Koller, A, Kaley, G

Oxidative stress contributes significantly to the development of cardiac pump dysfunction in hyperhomocysteinemia by Bagi, Z, Kaley, G, Koller, A

Transient elevation of glucose concentration reduces NO-mediation of flow-induced arteriolar dilation due to interaction between superoxide and tetrahydrobiopterin (BH4) by Bagi, Z, Koller, A, Kaley, G

Reduced sensitivity wall shear stress in arterioles of PECAM knockout mice due to impaired mechanotransduction by Bagi, Z, Frangos, J, Kaley, G

Enhanced superoxide production reduces NO-mediation of flow-dependent dilation of isolated coronary arteries in type 2 diabetes mellitus by Bagi, Z, Koller, A, Kaley, G

Upregulation of Arginase I Reduces Nitric Oxide-mediated Coronary Arteriolar Dilation in Patients With Type 2 Diabetes by Bagi, Z, Beleznai, T, Feher, A, Kaley, G

Microvascular dysfunction after transient high glucose is caused by superoxide-dependent reduction in the bioavailability of NO and BH(4). by Bagi, Z, Toth, E, Koller, A, Kaley, G

Role of platelet endothelial adhesion molecule (PECAM)-1 in sensing and mediating high temporal gradients of shear stress in arterioles by Koller, A, Frangos, J, Kaley, G, Bagi, Z

H2O2 increases production of constrictor prostaglandins in smooth muscle leading to enhanced arteriolar tone in Type 2 diabetic mice. by Erdei, N, Bagi, Z, Edes, I, Kaley, G, Koller, A

PECAM-1 mediates NO-dependent dilation of arterioles to high temporal gradients of shear stress. by Bagi, Z, Frangos, J, Yeh, J, White, C, Kaley, G, Koller, A

Exacerbation of endothelial dysfunction during the progression of diabetes: role of oxidative stress. by Huang, A, Yang, Y, Feher, A, Bagi, Z, Kaley, G, Sun, D

Hydrogen peroxide elicits COX-2-dependent production of constrictor prostaglandins in arterioles of type 2 diabetic mice by Bagi, Z, Erdei, N, Toth, A, Edes, I, Kaley, G, Koller, A

Type 2 diabetic mice have increased arteriolar tone and blood pressure: enhanced release of COX-2-derived constrictor prostaglandins. by Bagi, Z, Erdei, N, Toth, A, Li, W, Hintze, T, Koller, A, Kaley, G