HER2-positive breast cancer cells expressing elevated FAM83A are sensitive to FAM83A loss. by Courtney A Bartel, Mark W Jackson

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Targeting Pancreatic Cancer Cell Plasticity: The Latest in Therapeutics by Jacob M. Smigiel, Neetha Parameswaran, Mark W. Jackson

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HiJAK’d Signaling; the STAT3 Paradox in Senescence and Cancer Progression by Damian J. Junk, Benjamin L. Bryson, Mark W. Jackson

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Constitutive CCND1/CDK2 activity substitutes for p53 loss, or MYC or oncogenic RAS expression in the transformation of human mammary epithelial cells. by Damian J Junk, Rocky Cipriano, Martha Stampfer, Mark W Jackson

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Cancer Stem Cell Plasticity Drives Therapeutic Resistance by Mary R. Doherty, Jacob M. Smigiel, Damian J. Junk, Mark W. Jackson

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Balancing STAT Activity as a Therapeutic Strategy by Kelsey L. Polak, Noah M. Chernosky, Jacob M. Smigiel, Ilaria Tamagno, Mark W. Jackson

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Tumor Microenvironmental Signaling Elicits Epithelial-Mesenchymal Plasticity through Cooperation with Transforming Genetic Events by Damian J. Junk, Rocky Cipriano, Benjamin L. Bryson, Hannah L. Gilmore, Mark W. Jackson

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Green tea polyphenols induce p53-dependent and p53-independent apoptosis in prostate cancer cells through two distinct mechanisms. by Karishma Gupta, Vijay S Thakur, Natarajan Bhaskaran, Akbar Nawab, Melissa A Babcook, Mark W Jackson, Sanjay Gupta

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Inflammation mobilizes copper metabolism to promote colon tumorigenesis via an IL-17-STEAP4-XIAP axis by Yun Liao, Junjie Zhao, Katarzyna Bulek, Fangqiang Tang, Xing Chen, Gang Cai, Shang Jia, Paul L. Fox, Emina Huang, Theresa T. Pizarro, Matthew F. Kalady, Mark W. Jackson, Shideng Bao, Ganes C. Sen, George R. Stark, Christopher J. Chang, Xiaoxia Li

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