Showing 1 - 10 results of 10 for search 'Martin J. Allday', query time: 0.03s Refine Results
  1. 1
    Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression.

    Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression. by Jade Yee, Robert E White, Emma Anderton, Martin J Allday

    Published 2011-01-01
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  2. 2
    Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.

    Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP. by Lenka Skalska, Robert E White, Melanie Franz, Michaela Ruhmann, Martin J Allday

    Published 2010-06-01
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  3. 3
    Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

    Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2. by Quentin Bazot, Kostas Paschos, Lenka Skalska, Jens S Kalchschmidt, Gillian A Parker, Martin J Allday

    Published 2015-07-01
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  4. 4
    EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

    EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells. by Jens S Kalchschmidt, Adam C T Gillman, Kostas Paschos, Quentin Bazot, Bettina Kempkes, Martin J Allday

    Published 2016-01-01
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  5. 5
    Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification.

    Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification. by Robert E White, Ian J Groves, Ernest Turro, Jade Yee, Elisabeth Kremmer, Martin J Allday

    Published 2010-11-01
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  6. 6
    Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim.

    Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim. by Kostas Paschos, Paul Smith, Emma Anderton, Jaap M Middeldorp, Robert E White, Martin J Allday

    Published 2009-06-01
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  7. 7
    EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

    EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency. by Christine T Styles, Quentin Bazot, Gillian A Parker, Robert E White, Kostas Paschos, Martin J Allday

    Published 2017-08-01
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  8. 8
    Correction: Induction of p16 Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines.

    Correction: Induction of p16 Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines. by Lenka Skalska, Robert E. White, Gillian A. Parker, Ernest Turro, Alison J. Sinclair, Kostas Paschos, Martin J. Allday

    Published 2013-03-01
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  9. 9
    Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

    Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines. by Lenka Skalska, Robert E White, Gillian A Parker, Ernest Turro, Alison J Sinclair, Kostas Paschos, Martin J Allday

    Published 2013-02-01
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  10. 10
    Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection

    Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection by Alexander M Price, Joanne Dai, Quentin Bazot, Luv Patel, Pavel A Nikitin, Reza Djavadian, Peter S Winter, Cristina A Salinas, Ashley Perkins Barry, Kris C Wood, Eric C Johannsen, Anthony Letai, Martin J Allday, Micah A Luftig

    Published 2017-04-01
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