Transmitted HIV-1 is more virulent in heterosexual individuals than men-who-have-sex-with-men. by Ananthu James, Narendra M Dixit

Get full text

Mathematical model of viral kinetics in vitro estimates the number of E2-CD81 complexes necessary for hepatitis C virus entry. by Pranesh Padmanabhan, Narendra M Dixit

Get full text

Pre-existing resistance in the latent reservoir can compromise VRC01 therapy during chronic HIV-1 infection. by Ananya Saha, Narendra M Dixit

Get full text

Trade-off between synergy and efficacy in combinations of HIV-1 latency-reversing agents. by Vipul Gupta, Narendra M Dixit

Get full text

Timing the emergence of resistance to anti-HIV drugs with large genetic barriers. by Pankhuri Arora, Narendra M Dixit

Get full text

Viral kinetics suggests a reconciliation of the disparate observations of the modulation of claudin-1 expression on cells exposed to hepatitis C virus. by Pranesh Padmanabhan, Narendra M Dixit

Get full text

Transmitted HIV-1 is more virulent in heterosexual individuals than men-who-have-sex-with-men by Ananthu James, Narendra M. Dixit

Get full text

Increased B Cell Selection Stringency In Germinal Centers Can Explain Improved COVID-19 Vaccine Efficacies With Low Dose Prime or Delayed Boost by Amar K. Garg, Soumya Mittal, Pranesh Padmanabhan, Rajat Desikan, Narendra M. Dixit, Narendra M. Dixit

Get full text

Estimating the threshold surface density of Gp120-CCR5 complexes necessary for HIV-1 envelope-mediated cell-cell fusion. by Shiva Naresh Mulampaka, Narendra M Dixit

Get full text

Early exposure to broadly neutralizing antibodies may trigger a dynamical switch from progressive disease to lasting control of SHIV infection. by Rajat Desikan, Rubesh Raja, Narendra M Dixit

Get full text

Targeting TMPRSS2 and Cathepsin B/L together may be synergistic against SARS-CoV-2 infection. by Pranesh Padmanabhan, Rajat Desikan, Narendra M Dixit

Get full text

Preferential Presentation of High-Affinity Immune Complexes in Germinal Centers Can Explain How Passive Immunization Improves the Humoral Response by Amar K. Garg, Rajat Desikan, Narendra M. Dixit

Get full text

Modeling recapitulates the heterogeneous outcomes of SARS-CoV-2 infection and quantifies the differences in the innate immune and CD8 T-cell responses between patients experiencing... by Budhaditya Chatterjee, Harshbir Singh Sandhu, Narendra M Dixit

Get full text

Activation of Bacterial Histidine Kinases: Insights into the Kinetics of the <italic toggle="yes">cis</italic> Autophosphorylation Mechanism by Gaurav D. Sankhe, Narendra M. Dixit, Deepak K. Saini

Get full text

Modelling how responsiveness to interferon improves interferon-free treatment of hepatitis C virus infection. by Vishnu Venugopal, Pranesh Padmanabhan, Rubesh Raja, Narendra M Dixit

Get full text

A Low-Prevalence Single-Nucleotide Polymorphism in the Sensor Kinase PhoR in Mycobacterium tuberculosis Suppresses Its Autophosphatase Activity and Reduces Pathogenic Fitness: Impl... by Uchenna Watson Waturuocha, Uchenna Watson Waturuocha, M. S. Krishna, Vandana Malhotra, Narendra M. Dixit, Narendra M. Dixit, Deepak Kumar Saini, Deepak Kumar Saini

Get full text

Taking multiple infections of cells and recombination into account leads to small within-host effective-population-size estimates of HIV-1. by Rajesh Balagam, Vasantika Singh, Aparna Raju Sagi, Narendra M Dixit

Get full text

Influence of recombination on acquisition and reversion of immune escape and compensatory mutations in HIV-1 by Pradeep Nagaraja, Helen K. Alexander, Sebastian Bonhoeffer, Narendra M. Dixit

Get full text

Stochastic simulations suggest that HIV-1 survives close to its error threshold. by Kushal Tripathi, Rajesh Balagam, Nisheeth K Vishnoi, Narendra M Dixit

Get full text

An Evolutionary Paradigm Favoring Cross Talk between Bacterial Two-Component Signaling Systems by Bharadwaj Vemparala, Arjun Valiya Parambathu, Deepak Kumar Saini, Narendra M. Dixit

Get full text