Philip Hieter

Philip Hieter (born in 1952) is an American scientist specializing in yeast genetics. He is currently a professor of medical genetics at the Michael Smith Laboratories at the University of British Columbia. He is a member of the National Academy of Sciences. Provided by Wikipedia
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  1. 1
    Genome-wide profiling of yeast DNA:RNA hybrid prone sites with DRIP-chip.

    Genome-wide profiling of yeast DNA:RNA hybrid prone sites with DRIP-chip. by Yujia A Chan, Maria J Aristizabal, Phoebe Y T Lu, Zongli Luo, Akil Hamza, Michael S Kobor, Peter C Stirling, Philip Hieter

    Published 2014-04-01
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  2. 2
    An evolutionarily conserved synthetic lethal interaction network identifies FEN1 as a broad-spectrum target for anticancer therapeutic development.

    An evolutionarily conserved synthetic lethal interaction network identifies FEN1 as a broad-spectrum target for anticancer therapeutic development. by Derek M van Pel, Irene J Barrett, Yoko Shimizu, Babu V Sajesh, Brent J Guppy, Tom Pfeifer, Kirk J McManus, Philip Hieter

    Published 2013-01-01
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  3. 3
    The complete spectrum of yeast chromosome instability genes identifies candidate CIN cancer genes and functional roles for ASTRA complex components.

    The complete spectrum of yeast chromosome instability genes identifies candidate CIN cancer genes and functional roles for ASTRA complex components. by Peter C Stirling, Michelle S Bloom, Tejomayee Solanki-Patil, Stephanie Smith, Payal Sipahimalani, Zhijian Li, Megan Kofoed, Shay Ben-Aroya, Kyungjae Myung, Philip Hieter

    Published 2011-04-01
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  4. 4
    Synthetic lethality of cohesins with PARPs and replication fork mediators.

    Synthetic lethality of cohesins with PARPs and replication fork mediators. by Jessica L McLellan, Nigel J O'Neil, Irene Barrett, Elizabeth Ferree, Derek M van Pel, Kevin Ushey, Payal Sipahimalani, Jennifer Bryan, Ann M Rose, Philip Hieter

    Published 2012-01-01
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  5. 5
    Predisposition to cancer caused by genetic and functional defects of mammalian Atad5.

    Predisposition to cancer caused by genetic and functional defects of mammalian Atad5. by Daphne W Bell, Nilabja Sikdar, Kyoo-Young Lee, Jessica C Price, Raghunath Chatterjee, Hee-Dong Park, Jennifer Fox, Masamichi Ishiai, Meghan L Rudd, Lana M Pollock, Sarah K Fogoros, Hassan Mohamed, Christin L Hanigan, NISC Comparative Sequencing Program, Suiyuan Zhang, Pedro Cruz, Gabriel Renaud, Nancy F Hansen, Praveen F Cherukuri, Bhavesh Borate, Kirk J McManus, Jan Stoepel, Payal Sipahimalani, Andrew K Godwin, Dennis C Sgroi, Maria J Merino, Gene Elliot, Abdel Elkahloun, Charles Vinson, Minoru Takata, James C Mullikin, Tyra G Wolfsberg, Philip Hieter, Dae-Sik Lim, Kyungjae Myung

    Published 2011-08-01
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