Richard Kolodner

Richard David Kolodner is an American scientist with Ludwig Cancer Research who has made research contributions to the genetic basis for inherited susceptibility to common cancers. He is a Distinguished Professor of Cellular and Molecular Medicine at the UC San Diego School of Medicine. Kolodner is a member of the National Academy of Sciences and National Academy of Medicine, and he is a fellow of the American Academy of Arts and Sciences. Provided by Wikipedia
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  1. 1
    Stabilization of dicentric translocations through secondary rearrangements mediated by multiple mechanisms in S. cerevisiae.

    Stabilization of dicentric translocations through secondary rearrangements mediated by multiple mechanisms in S. cerevisiae. by Vincent Pennaneach, Richard D Kolodner

    Published 2009-07-01
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  2. 2
    Rapid analysis of Saccharomyces cerevisiae genome rearrangements by multiplex ligation-dependent probe amplification.

    Rapid analysis of Saccharomyces cerevisiae genome rearrangements by multiplex ligation-dependent probe amplification. by Jason E Chan, Richard D Kolodner

    Published 2012-01-01
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  3. 3
    A genetic and structural study of genome rearrangements mediated by high copy repeat Ty1 elements.

    A genetic and structural study of genome rearrangements mediated by high copy repeat Ty1 elements. by Jason E Chan, Richard D Kolodner

    Published 2011-05-01
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  4. 4
    Rad5 and Its Human Homologs, HLTF and SHPRH, Are Novel Interactors of Mismatch Repair

    Rad5 and Its Human Homologs, HLTF and SHPRH, Are Novel Interactors of Mismatch Repair by Anna K. Miller, Guogen Mao, Breanna G. Knicely, Hannah G. Daniels, Christine Rahal, Christopher D. Putnam, Christopher D. Putnam, Richard D. Kolodner, Richard D. Kolodner, Richard D. Kolodner, Richard D. Kolodner, Eva M. Goellner, Eva M. Goellner

    Published 2022-06-01
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  5. 5
    Post-replication repair suppresses duplication-mediated genome instability.

    Post-replication repair suppresses duplication-mediated genome instability. by Christopher D Putnam, Tikvah K Hayes, Richard D Kolodner

    Published 2010-05-01
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  6. 6
    Checkpoint Kinases Regulate a Global Network of Transcription Factors in Response to DNA Damage

    Checkpoint Kinases Regulate a Global Network of Transcription Factors in Response to DNA Damage by Eric J. Jaehnig, Dwight Kuo, Hans Hombauer, Trey G. Ideker, Richard D. Kolodner

    Published 2013-07-01
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  7. 7
    The Swr1 chromatin-remodeling complex prevents genome instability induced by replication fork progression defects

    The Swr1 chromatin-remodeling complex prevents genome instability induced by replication fork progression defects by Anjana Srivatsan, Bin-Zhong Li, Barnabas Szakal, Dana Branzei, Christopher D. Putnam, Richard D. Kolodner

    Published 2018-09-01
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  8. 8
    Correction: Distinct SUMO Ligases Cooperate with Esc2 and Slx5 to Suppress Duplication-Mediated Genome Rearrangements.

    Correction: Distinct SUMO Ligases Cooperate with Esc2 and Slx5 to Suppress Duplication-Mediated Genome Rearrangements. by Claudio P Albuquerque, Guoliang Wang, Nancy S Lee, Richard D Kolodner, Christopher D Putnam, Huilin Zhou

    Published 2016-08-01
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  9. 9
    SUMO E3 ligase Mms21 prevents spontaneous DNA damage induced genome rearrangements.

    SUMO E3 ligase Mms21 prevents spontaneous DNA damage induced genome rearrangements. by Jason Liang, Bin-Zhong Li, Alexander P Tan, Richard D Kolodner, Christopher D Putnam, Huilin Zhou

    Published 2018-03-01
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  10. 10
    Peroxiredoxin Tsa1 is the key peroxidase suppressing genome instability and protecting against cell death in Saccharomyces cerevisiae.

    Peroxiredoxin Tsa1 is the key peroxidase suppressing genome instability and protecting against cell death in Saccharomyces cerevisiae. by Ismail Iraqui, Guy Kienda, Jérémie Soeur, Gérard Faye, Giuseppe Baldacci, Richard D Kolodner, Meng-Er Huang

    Published 2009-06-01
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  11. 11
    Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an exonuclease 1-independent mismatch repair pathway.

    Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an exonuclease 1-independent mismatch repair pathway. by Catherine E Smith, Marc L Mendillo, Nikki Bowen, Hans Hombauer, Christopher S Campbell, Arshad Desai, Christopher D Putnam, Richard D Kolodner

    Published 2013-10-01
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  12. 12
    Cdc73 suppresses genome instability by mediating telomere homeostasis.

    Cdc73 suppresses genome instability by mediating telomere homeostasis. by Rahul V Nene, Christopher D Putnam, Bin-Zhong Li, Katarina G Nguyen, Anjana Srivatsan, Christopher S Campbell, Arshad Desai, Richard D Kolodner

    Published 2018-01-01
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  13. 13
    Loss of the thioredoxin reductase Trr1 suppresses the genomic instability of peroxiredoxin tsa1 mutants.

    Loss of the thioredoxin reductase Trr1 suppresses the genomic instability of peroxiredoxin tsa1 mutants. by Sandrine Ragu, Michèle Dardalhon, Sushma Sharma, Ismail Iraqui, Géraldine Buhagiar-Labarchède, Virginie Grondin, Guy Kienda, Laurence Vernis, Roland Chanet, Richard D Kolodner, Meng-Er Huang, Gérard Faye

    Published 2014-01-01
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