Showing 1 - 5 results of 5 for search 'Saurav De', query time: 0.03s Refine Results
  1. 1
    RNAi Transfection Optimized in Primary Naïve B Cells for the Targeted Analysis of Human Plasma Cell Differentiation

    RNAi Transfection Optimized in Primary Naïve B Cells for the Targeted Analysis of Human Plasma Cell Differentiation by Tiffany Shih, Saurav De, Saurav De, Betsy J. Barnes, Betsy J. Barnes

    Published 2019-07-01
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  2. 2
    Distinct fibroblast functions associated with fibrotic and immune-mediated inflammatory diseases and their implications for therapeutic development [version 1; peer review: 1 approved, 2 approved with reservations]

    Distinct fibroblast functions associated with fibrotic and immune-mediated inflammatory diseases and their implications for therapeutic development [version 1; peer review: 1 appro... by Saurav De, Alexander M. S. Barron, Thomas Fabre

    Published 2024-01-01
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  3. 3
    B Cell-Intrinsic Role for IRF5 in TLR9/BCR-Induced Human B Cell Activation, Proliferation, and Plasmablast Differentiation

    B Cell-Intrinsic Role for IRF5 in TLR9/BCR-Induced Human B Cell Activation, Proliferation, and Plasmablast Differentiation by Saurav De, Saurav De, Baohong Zhang, Tiffany Shih, Sukhwinder Singh, Aaron Winkler, Robert Donnelly, Betsy J. Barnes, Betsy J. Barnes

    Published 2018-01-01
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  4. 4
    Chemical genetics reveals a specific requirement for Cdk2 activity in the DNA damage response and identifies Nbs1 as a Cdk2 substrate in human cells.

    Chemical genetics reveals a specific requirement for Cdk2 activity in the DNA damage response and identifies Nbs1 as a Cdk2 substrate in human cells. by Lara Wohlbold, Karl A Merrick, Saurav De, Ramon Amat, Jun Hyun Kim, Stéphane Larochelle, Jasmina J Allen, Chao Zhang, Kevan M Shokat, John H J Petrini, Robert P Fisher

    Published 2012-08-01
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  5. 5
    Loss of ATRX, genome instability, and an altered DNA damage response are hallmarks of the alternative lengthening of telomeres pathway.

    Loss of ATRX, genome instability, and an altered DNA damage response are hallmarks of the alternative lengthening of telomeres pathway. by Courtney A Lovejoy, Wendi Li, Steven Reisenweber, Supawat Thongthip, Joanne Bruno, Titia de Lange, Saurav De, John H J Petrini, Patricia A Sung, Maria Jasin, Joseph Rosenbluh, Yaara Zwang, Barbara A Weir, Charlie Hatton, Elena Ivanova, Laura Macconaill, Megan Hanna, William C Hahn, Neal F Lue, Roger R Reddel, Yuchen Jiao, Kenneth Kinzler, Bert Vogelstein, Nickolas Papadopoulos, Alan K Meeker, ALT Starr Cancer Consortium

    Published 2012-01-01
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