Pinpointing Synaptic Loss Caused by Alzheimer’s Disease with fMRI 由 Adam M. Brickman, Scott A. Small, Adam Fleisher

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A deep learning MRI approach outperforms other biomarkers of prodromal Alzheimer’s disease 由 Xinyang Feng, Frank A. Provenzano, Scott A. Small, for the Alzheimer’s Disease Neuroimaging Initiative

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Deep learning of MRI contrast enhancement for mapping cerebral blood volume from single-modal non-contrast scans of aging and Alzheimer's disease brains 由 Chen Liu, Nanyan Zhu, Haoran Sun, Junhao Zhang, Xinyang Feng, Sabrina Gjerswold-Selleck, Dipika Sikka, Xuemin Zhu, Xueqing Liu, Tal Nuriel, Hong-Jian Wei, Cheng-Chia Wu, J. Thomas Vaughan, Andrew F. Laine, Frank A. Provenzano, Scott A. Small, Scott A. Small, Scott A. Small, Jia Guo, Jia Guo

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The location and trafficking routes of the neuronal retromer and its role in amyloid precursor protein transport 由 Akhil Bhalla, Christopher P. Vetanovetz, Etienne Morel, Zeina Chamoun, Gilbert Di Paolo, Scott A. Small

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Hyperleucinemia causes hippocampal retromer deficiency linking diabetes to Alzheimer's disease 由 Michael V. Morabito, Diego E. Berman, Remy T. Schneider, Yiying Zhang, Rudolph L. Leibel, Scott A. Small

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Temporal lobe epilepsy lateralization using retrospective cerebral blood volume MRI 由 Xinyang Feng, Marla J. Hamberger, Hannah C. Sigmon, Jia Guo, Scott A. Small, Frank A. Provenzano

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Trans-synaptic spread of tau pathology in vivo. 由 Li Liu, Valerie Drouet, Jessica W Wu, Menno P Witter, Scott A Small, Catherine Clelland, Karen Duff

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Stabilizing the Retromer Complex in a Human Stem Cell Model of Alzheimer’s Disease Reduces TAU Phosphorylation Independently of Amyloid Precursor Protein 由 Jessica E. Young, Lauren K. Fong, Harald Frankowski, Gregory A. Petsko, Scott A. Small, Lawrence S.B. Goldstein

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Brain regions vulnerable and resistant to aging without Alzheimer's disease. 由 Xinyang Feng, Jia Guo, Hannah C Sigmon, Richard P Sloan, Adam M Brickman, Frank A Provenzano, Scott A Small, Alzheimer’s Disease Neuroimaging Initiative

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Retromer deficiency in Tauopathy models enhances the truncation and toxicity of Tau 由 Jamshid Asadzadeh, Evelyne Ruchti, Wei Jiao, Greta Limoni, Catherine MacLachlan, Scott A. Small, Graham Knott, Ismael Santa-Maria, Brian D. McCabe

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Insights into the role of diet and dietary flavanols in cognitive aging: results of a randomized controlled trial 由 Richard P. Sloan, Melanie Wall, Lok-Kin Yeung, Tianshu Feng, Xinyang Feng, Frank Provenzano, Hagen Schroeter, Vincenzo Lauriola, Adam M. Brickman, Scott A. Small

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Depletion of the AD Risk Gene SORL1 Selectively Impairs Neuronal Endosomal Traffic Independent of Amyloidogenic APP Processing 由 Allison Knupp, Swati Mishra, Refugio Martinez, Jacquelyn E. Braggin, Marcell Szabo, Chizuru Kinoshita, Dale W. Hailey, Scott A. Small, Suman Jayadev, Jessica E. Young

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Detecting schizophrenia with 3D structural brain MRI using deep learning 由 Junhao Zhang, Vishwanatha M. Rao, Ye Tian, Yanting Yang, Nicolas Acosta, Zihan Wan, Pin-Yu Lee, Chloe Zhang, Lawrence S. Kegeles, Scott A. Small, Jia Guo

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Neuronal lysosomal dysfunction releases exosomes harboring APP C-terminal fragments and unique lipid signatures 由 André M. Miranda, Zofia M. Lasiecka, Yimeng Xu, Jessi Neufeld, Sanjid Shahriar, Sabrina Simoes, Robin B. Chan, Tiago Gil Oliveira, Scott A. Small, Gilbert Di Paolo

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Corrigendum to “Regional vulnerability in Huntington's disease: fMRI-guided molecular analysis in patients and a mouse model of disease” [Neurobiol Dis 52 (2013) 84–93] 由 Nicole M. Lewandowski, Yvette Bordelon, Adam M. Brickman, Sergio Angulo, Usman Khan, Jordan Muraskin, Erica Y. Griffith, Paula Wasserman, Liliana Menalled, Jean Paul Vonsattel, Karen Marder, Scott A. Small, Herman Moreno

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Regional vulnerability in Huntington's disease: fMRI-guided molecular analysis in patients and a mouse model of disease 由 Nicole M. Lewandowski, Yvette Bordelon, Adam M. Brickman, Sergio Angulo, Usman Khan, Jordan Muraskin, Erica Y. Griffith, Paula Wasserman, Liliana Menalled, Jean Paul Vonsattel, Karen Marder, Scott A. Small, Herman Moreno

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Neuronal hyperactivity due to loss of inhibitory tone in APOE4 mice lacking Alzheimer’s disease-like pathology 由 Tal Nuriel, Sergio L. Angulo, Usman Khan, Archana Ashok, Qiuying Chen, Helen Y. Figueroa, Sheina Emrani, Li Liu, Mathieu Herman, Geoffrey Barrett, Valerie Savage, Luna Buitrago, Efrain Cepeda-Prado, Christine Fung, Eliana Goldberg, Steven S. Gross, S. Abid Hussaini, Herman Moreno, Scott A. Small, Karen E. Duff

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