Corrigendum to “Endoplasmic reticulum stress is activated in post-ischemic kidneys to promote chronic kidney disease” [EBioMedicine 37 (2018) 269–280] by Shaoqun Shu, Jiefu Zhu, Zhiwen Liu, Chengyuan Tang, Juan Cai, Zheng Dong

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Endoplasmic reticulum stress is activated in post-ischemic kidneys to promote chronic kidney diseaseResearch in context by Shaoqun Shu, Jiefu Zhu, Zhiwen Liu, Chengyuan Tang, Juan Cai, Zheng Dong

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Hypoxia and Hypoxia-Inducible Factors in Kidney Injury and Repair by Shaoqun Shu, Ying Wang, Meiling Zheng, Zhiwen Liu, Juan Cai, Chengyuan Tang, Zheng Dong

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AMPK/mTOR Signaling in Autophagy Regulation During Cisplatin-Induced Acute Kidney Injury by Ying Wang, Zhiwen Liu, Shaoqun Shu, Juan Cai, Chengyuan Tang, Zheng Dong, Zheng Dong

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Red Blood Cell Distribution Width and Neutrophil-to-Lymphocyte Ratio in Predicting Adverse Outcomes of Acute Kidney Injury in Hospitalized Patients by Jiefu Zhu, Cong Zeng, Lei Zhang, Shaoqun Shu, Yinghong Liu, Guochun Chen, Hong Liu, Yu Liu, Zheng Dong

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Reciprocal regulation between ER stress and autophagy in renal tubular fibrosis and apoptosis by Shaoqun Shu, Hui Wang, Jiefu Zhu, Zhiwen Liu, Danyi Yang, Wenwen Wu, Juan Cai, Anqun Chen, Chengyuan Tang, Zheng Dong

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The PINK1/PARK2/optineurin pathway of mitophagy is activated for protection in septic acute kidney injury by Ying Wang, Jiefu Zhu, Zhiwen Liu, Shaoqun Shu, Ying Fu, Yuxue Liu, Juan Cai, Chengyuan Tang, Yu Liu, Xiaoming Yin, Zheng Dong

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