Impaired exercise capacity in cytosolic (M-CK) but not in mitochondrial creatine kinase knockout mice 由 Maass, A, Bauer, L, Neubauer, S, Spindler, M

Mitochondrial creatine kinase is critically necessary to maintain a normal profile of myocardial high-energy phosphates 由 Spindler, M, Niebler, A, Horn, M, Lanz, T, Schnackerz, K, Neubauer, S

Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism. 由 Spindler, M, Niebler, R, Remkes, H, Horn, M, Lanz, T, Neubauer, S

Altered calcium homeostasis and increased susceptibility to ischemia in creatine kinase-deficient hearts 由 Spindler, M, Meyer, K, Leupold, A, Wagner, H, Stromer, H, Neubauer, S

Interference of angiotensin-converting enzyme inhibition with vasoactive peptides in the coronary circulation of dogs. 由 Bauer, B, Neubauer, S, Spindler, M, Dürr, R, Becker, H, Ertl, G

Alterations in the myocardial creatine kinase system precede the development of contractile dysfunction in beta(1)-adrenergic receptor transgenic mice. 由 Spindler, M, Engelhardt, S, Niebler, R, Wagner, H, Hein, L, Lohse, M, Neubauer, S

Temporal fluctuations of myocardia high-energy phosphate metabolite with the cardiac cycle. 由 Spindler, M, Illing, B, Horn, M, de Groot, M, Ertl, G, Neubauer, S

Functional assessment of isolated right heart failure by high resolution in-vivo cardiovascular magnetic resonance in mice. 由 Frydrychowicz, A, Spindler, M, Rommel, E, Ertl, G, Haase, A, Neubauer, S, Wiesmann, F

Creatine kinase-deficient hearts exhibit increased susceptibility to ischemia-reperfusion injury and impaired calcium homeostasis. 由 Spindler, M, Meyer, K, Strömer, H, Leupold, A, Boehm, E, Wagner, H, Neubauer, S

Left-ventricular hypertrophy and post-infarct remodelling in mice lacking creatine kinase isoenzyme assessed by magnetic resonance imaging 由 Nahrendorf, M, Hu, K, Hiller, K, Nordbeck, P, Neubauer, S, Ertl, G, Bauer, W, Spindler, M

Downregulation of the Na(+)-creatine cotransporter in failing human myocardium and in experimental heart failure. 由 Neubauer, S, Remkes, H, Spindler, M, Horn, M, Wiesmann, F, Prestle, J, Walzel, B, Ertl, G, Hasenfuss, G, Wallimann, T

Creatine kinase knockout mice show left ventricular hypertrophy and dilatation, but unaltered remodeling post-myocardial infarction. 由 Nahrendorf, M, Spindler, M, Hu, K, Bauer, L, Ritter, O, Nordbeck, P, Quaschning, T, Hiller, K, Wallis, J, Ertl, G, Bauer, W, Neubauer, S

Multimodal functional cardiac MRI in creatine kinase-deficient mice reveals subtle abnormalities in myocardial perfusion and mechanics. 由 Nahrendorf, M, Streif, J, Hiller, K, Hu, K, Nordbeck, P, Ritter, O, Sosnovik, D, Bauer, L, Neubauer, S, Jakob, P, Ertl, G, Spindler, M, Bauer, W

alpha-Tropomyosin mutations Asp(175)Asn and Glu(180)Gly affect cardiac function in transgenic rats in different ways. 由 Wernicke, D, Thiel, C, Duja-Isac, C, Essin, K, Spindler, M, Nunez, D, Plehm, R, Wessel, N, Hammes, A, Edwards, R, Lippoldt, A, Zacharias, U, Strömer, H, Neubauer, S, Davies, M, Morano, I, Thierfelder, L