Seneca Valley Virus 3Cpro Mediates Cleavage and Redistribution of Nucleolin To Facilitate Viral Replication
ABSTRACT Seneca Valley virus (SVV) is a recently discovered pathogen that poses a significant threat to the global pig industry. It has been shown that many viruses are reliant on nucleocytoplasmic trafficking of nucleolin (NCL) for their own replication. Here, we demonstrate that NCL, a critical pr...
Main Authors: | Jiangwei Song, Rong Quan, Dan Wang, Jue Liu |
---|---|
Format: | Article |
Language: | English |
Published: |
American Society for Microbiology
2022-04-01
|
Series: | Microbiology Spectrum |
Subjects: | |
Online Access: | https://journals.asm.org/doi/10.1128/spectrum.00304-22 |
Similar Items
-
Seneca Valley Virus 2C and 3Cpro Induce Apoptosis via Mitochondrion-Mediated Intrinsic Pathway
by: Tingting Liu, et al.
Published: (2019-05-01) -
Seneca Valley Virus 3Cpro Cleaves Heterogeneous Nuclear Ribonucleoprotein K to Facilitate Viral Replication
by: Jiangwei Song, et al.
Published: (2022-07-01) -
Pathogenicity of Seneca Valley virus in pigs and detection in Culicoides from an infected pig farm
by: Jinyong Zhang, et al.
Published: (2021-10-01) -
Seneca Valley virus replicons are packaged in trans and have the capacity to overcome the limitations of viral transgene expression
by: Jeffrey D. Bryant, et al.
Published: (2023-03-01) -
Dexamethasone treatment did not exacerbate Seneca Valley virus infection in nursery-age pigs
by: Alexandra Buckley, et al.
Published: (2018-11-01)