WIP Modulates Oxidative Stress through NRF2/KEAP1 in Glioblastoma Cells
Due to their high metabolic rate, tumor cells produce exacerbated levels of reactive oxygen species that need to be under control. Wiskott–Aldrich syndrome protein (WASP)-interacting protein (WIP) is a scaffold protein with multiple yet poorly understood functions that participates in tumor progress...
Main Authors: | Maribel Escoll, Diego Lastra, Natalia Robledinos-Antón, Francisco Wandosell, Inés María Antón, Antonio Cuadrado |
---|---|
Format: | Article |
Language: | English |
Published: |
MDPI AG
2020-08-01
|
Series: | Antioxidants |
Subjects: | |
Online Access: | https://www.mdpi.com/2076-3921/9/9/773 |
Similar Items
-
WIP, YAP/TAZ and Actin Connections Orchestrate Development and Transformation in the Central Nervous System
by: Inés M. Antón, et al.
Published: (2021-06-01) -
Transcription factor NRF2 uses the Hippo pathway effector TAZ to induce tumorigenesis in glioblastomas
by: Maribel Escoll, et al.
Published: (2020-02-01) -
Transcription Factor NRF2 Participates in Cell Cycle Progression at the Level of G1/S and Mitotic Checkpoints
by: Diego Lastra, et al.
Published: (2022-05-01) -
TAZ Represses the Neuronal Commitment of Neural Stem Cells
by: Natalia Robledinos-Antón, et al.
Published: (2020-10-01) -
WIP-YAP/TAZ as A New Pro-Oncogenic Pathway in Glioma
by: Sergio Rivas, et al.
Published: (2018-06-01)