INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS
Atherosclerotic cardiovascular diseases are the leading cause of death worldwide. In recent decades, the influence of many microorganisms involved (directly or indirectly) in provoking the process of atherogenesis, including cell adhesion, cytokine-associated damage, release of reactive oxygen speci...
| Main Authors: | , , |
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| Format: | Article |
| Language: | Russian |
| Published: |
Siberian Branch of Russian Academy of Sciences, Research Institute of Internal and Preventive Medicine, branch of the Institute of Cytology and Genetics
2019-06-01
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| Series: | Атеросклероз |
| Subjects: | |
| Online Access: | https://ateroskleroz.elpub.ru/jour/article/view/351/321 |
| _version_ | 1797963691021303808 |
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| author | O. V. Reshetnikov S. A. Kurilovich Yu. P. Nikitin |
| author_facet | O. V. Reshetnikov S. A. Kurilovich Yu. P. Nikitin |
| author_sort | O. V. Reshetnikov |
| collection | DOAJ |
| description | Atherosclerotic cardiovascular diseases are the leading cause of death worldwide. In recent decades, the influence of many microorganisms involved (directly or indirectly) in provoking the process of atherogenesis, including cell adhesion, cytokine-associated damage, release of reactive oxygen species, etc. has been shown. Others, such as the influenza virus, can cause systemic inflammation that can damage vascular wall (for example, by cytokines and proteases). In addition, another indirect mechanism by which some infectious agents (such as Helicobacter pylori, Chlamydia pneumoniae, periodontal pathogens, etc.) that may play a role in the pathogenesis of atherosclerosis is molecular mimicry. Given the complexity of the mechanisms by which each pathogen can contribute to the development and progression of atherosclerosis, it is obvious that ongoing research and new data will be useful for improving our understanding of the infectious component of atherosclerosis. |
| first_indexed | 2024-04-11T01:32:15Z |
| format | Article |
| id | doaj.art-1f213ac99198437baacc2ddc1a466e47 |
| institution | Directory Open Access Journal |
| issn | 2078-256X |
| language | Russian |
| last_indexed | 2024-04-11T01:32:15Z |
| publishDate | 2019-06-01 |
| publisher | Siberian Branch of Russian Academy of Sciences, Research Institute of Internal and Preventive Medicine, branch of the Institute of Cytology and Genetics |
| record_format | Article |
| series | Атеросклероз |
| spelling | doaj.art-1f213ac99198437baacc2ddc1a466e472023-01-03T09:22:10ZrusSiberian Branch of Russian Academy of Sciences, Research Institute of Internal and Preventive Medicine, branch of the Institute of Cytology and GeneticsАтеросклероз2078-256X2019-06-011527888https://doi.org/10.15372/ATER20190211INFECTIONS, INFLAMMATION AND ATHEROSCLEROSISO. V. Reshetnikov0S. A. Kurilovich1Yu. P. Nikitin2Research Institute of Internal and Preventive Medicine – Branch of Federal Research Institute of Cytology and Genetics of SB RAS Russian FederationResearch Institute of Internal and Preventive Medicine – Branch of Federal Research Institute of Cytology and Genetics of SB RAS; Novosibirsk State Medical University of Minzdrav of Russia Russian FederationResearch Institute of Internal and Preventive Medicine – Branch of Federal Research Institute of Cytology and Genetics of SB RAS Russian FederationAtherosclerotic cardiovascular diseases are the leading cause of death worldwide. In recent decades, the influence of many microorganisms involved (directly or indirectly) in provoking the process of atherogenesis, including cell adhesion, cytokine-associated damage, release of reactive oxygen species, etc. has been shown. Others, such as the influenza virus, can cause systemic inflammation that can damage vascular wall (for example, by cytokines and proteases). In addition, another indirect mechanism by which some infectious agents (such as Helicobacter pylori, Chlamydia pneumoniae, periodontal pathogens, etc.) that may play a role in the pathogenesis of atherosclerosis is molecular mimicry. Given the complexity of the mechanisms by which each pathogen can contribute to the development and progression of atherosclerosis, it is obvious that ongoing research and new data will be useful for improving our understanding of the infectious component of atherosclerosis.https://ateroskleroz.elpub.ru/jour/article/view/351/321atherosclerosisbacteriavirusespathogenetic mechanisms |
| spellingShingle | O. V. Reshetnikov S. A. Kurilovich Yu. P. Nikitin INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS Атеросклероз atherosclerosis bacteria viruses pathogenetic mechanisms |
| title | INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS |
| title_full | INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS |
| title_fullStr | INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS |
| title_full_unstemmed | INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS |
| title_short | INFECTIONS, INFLAMMATION AND ATHEROSCLEROSIS |
| title_sort | infections inflammation and atherosclerosis |
| topic | atherosclerosis bacteria viruses pathogenetic mechanisms |
| url | https://ateroskleroz.elpub.ru/jour/article/view/351/321 |
| work_keys_str_mv | AT ovreshetnikov infectionsinflammationandatherosclerosis AT sakurilovich infectionsinflammationandatherosclerosis AT yupnikitin infectionsinflammationandatherosclerosis |