IKCa Channels Are a Critical Determinant of the Slow AHP in CA1 Pyramidal Neurons
Control over the frequency and pattern of neuronal spike discharge depends on Ca2+-gated K+ channels that reduce cell excitability by hyperpolarizing the membrane potential. The Ca2+-dependent slow afterhyperpolarization (sAHP) is one of the most prominent inhibitory responses in the brain, with sAH...
Main Authors: | Brian King, Arsalan P. Rizwan, Hadhimulya Asmara, Norman C. Heath, Jordan D.T. Engbers, Steven Dykstra, Theodore M. Bartoletti, Shahid Hameed, Gerald W. Zamponi, Ray W. Turner |
---|---|
Format: | Article |
Language: | English |
Published: |
Elsevier
2015-04-01
|
Series: | Cell Reports |
Online Access: | http://www.sciencedirect.com/science/article/pii/S2211124715002958 |
Similar Items
-
A T-type channel-calmodulin complex triggers αCaMKII activation
by: Hadhimulya Asmara, et al.
Published: (2017-08-01) -
Low voltage activation of KCa1.1 current by Cav3-KCa1.1 complexes.
by: Renata Rehak, et al.
Published: (2013-01-01) -
Signal processing by T-type calcium channel interactions in the cerebellum
by: Jordan D.T. Engbers, et al.
Published: (2013-11-01) -
IKCa channels control breast cancer metabolism including AMPK-driven autophagy
by: Dominic Gross, et al.
Published: (2022-10-01) -
Differential roles for IKCa and SKCa channels in EDHF-type responses in mouse mesenteric arteries
by: Gallagher, N, et al.
Published: (2006)