Repeated administration of fenitrothion alters renal functions via oxidative stress mechanism without inhibiting acetylcholinesterase activity in rats

Repeated administration of fenitrothion alters renal functions via oxidative stress mechanism without inhibiting acetylcholinesterase activity in rats

Background: The main mechanism of fenitrothion (FNT) toxicity is by inhibiting the acetylcholinesterase enzyme, though studies have shown that FNT might also develop other toxicological manifestations, including oxidative stress. However, the kidney functions as well as oxidative stress status after...

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Bibliographic Details
Main Authors: Nur Afizah Yusoff, Izzah Irdina Juremi, Siti Balkis Budin, Izatus Shima Taib
Format: Article
Language:English
Published: Biome Scientia 2020-11-01
Series:Life Sciences, Medicine and Biomedicine
Subjects:
antioxidant
non-amyloid
oxidative stress
organophosphate
oxidative damage
Online Access:http://biomescientia.com/index.php/lsmb/article/view/70

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