Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis
<p>Abstract</p> <p>Background</p> <p>Myelin Oligodendrocyte Glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE) is the most commonly used mouse model for multiple sclerosis (MS). During the of progression of EAE, microglia, the immunocompetent cel...
Main Authors: | Tsirka Stella E, Wu Muzhou, Bhasin Madhuri |
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Format: | Article |
Language: | English |
Published: |
BMC
2007-07-01
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Series: | BMC Immunology |
Online Access: | http://www.biomedcentral.com/1471-2172/8/10 |
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