Author Correction: Generation of G51D and 3D mice reveals decreased α-synuclein tetramer-monomer ratios promote Parkinson’s disease phenotypes
Main Authors: | Silke Nuber, Xiaoqun Zhang, Thomas D. McCaffery, Tim E. Moors, Marie-Alexandre Adom, Wolf N. Hahn, Dylan Martin, Maria Ericsson, Arati Tripathi, Ulf Dettmer, Per Svenningsson, Dennis J. Selkoe |
---|---|
Format: | Article |
Language: | English |
Published: |
Nature Portfolio
2024-03-01
|
Series: | npj Parkinson's Disease |
Online Access: | https://doi.org/10.1038/s41531-024-00675-5 |
Similar Items
-
Generation of G51D and 3D mice reveals decreased α-synuclein tetramer-monomer ratios promote Parkinson’s disease phenotypes
by: Silke Nuber, et al.
Published: (2024-02-01) -
Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation
by: Dettmer, Ulf, et al.
Published: (2015) -
Reducing the lipase LIPE in mutant α-synuclein mice improves Parkinson-like deficits and reveals sex differences in fatty acid metabolism
by: M.A. Adom, et al.
Published: (2024-09-01) -
Lipotoxicity Downstream of α-Synuclein Imbalance: A Relevant Pathomechanism in Synucleinopathies?
by: Arati Tripathi, et al.
Published: (2021-12-01) -
A new method for quantitative immunoblotting of endogenous α-synuclein.
by: Andrew J Newman, et al.
Published: (2013-01-01)