Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria
The deposition of Aβ42 has been regarded as one of the important pathological features of Alzheimer’s disease (AD). However, drug development for Aβ42 toxicity has been progressed slowly.
| Main Authors: | , , , , , , |
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| Format: | Article |
| Language: | English |
| Published: |
De Gruyter
2024-04-01
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| Series: | Translational Neuroscience |
| Subjects: | |
| Online Access: | https://doi.org/10.1515/tnsci-2022-0338 |
| _version_ | 1797208423275692032 |
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| author | Li Liangxian Huang Zhiheng Wu Mingli Li Xia Xiao Bo Yao Dong Mo Biwen |
| author_facet | Li Liangxian Huang Zhiheng Wu Mingli Li Xia Xiao Bo Yao Dong Mo Biwen |
| author_sort | Li Liangxian |
| collection | DOAJ |
| description | The deposition of Aβ42 has been regarded as one of the important pathological features of Alzheimer’s disease (AD). However, drug development for Aβ42 toxicity has been progressed slowly. |
| first_indexed | 2024-04-24T09:38:34Z |
| format | Article |
| id | doaj.art-3d12c668caaf4e95b3747997f5310b0a |
| institution | Directory Open Access Journal |
| issn | 2081-6936 |
| language | English |
| last_indexed | 2024-04-24T09:38:34Z |
| publishDate | 2024-04-01 |
| publisher | De Gruyter |
| record_format | Article |
| series | Translational Neuroscience |
| spelling | doaj.art-3d12c668caaf4e95b3747997f5310b0a2024-04-15T07:42:36ZengDe GruyterTranslational Neuroscience2081-69362024-04-011515051710.1515/tnsci-2022-0338Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondriaLi Liangxian0Huang Zhiheng1Wu Mingli2Li Xia3Xiao Bo4Yao Dong5Mo Biwen6Department of Respiratory and Critical Care Medicine, The Second Affiliated Hospital of Guilin Medical University, Guilin, 541199, ChinaDepartment of Respiratory and Critical Care Medicine, The Second Affiliated Hospital of Guilin Medical University, Guilin, 541199, ChinaGuangxi Key Laboratory of Brain and Cognitive Neuroscience, Guilin Medical University, Guilin, 541199, ChinaGuangxi Key Laboratory of Brain and Cognitive Neuroscience, Guilin Medical University, Guilin, 541199, ChinaLaboratory of Respiratory Disease, Affiliated Hospital of Guilin Medical University, Guilin, 541002, ChinaDepartment of Respiratory and Critical Care Medicine, The Second Affiliated Hospital of Guilin Medical University, Guilin, 541199, ChinaDepartment of Respiratory and Critical Care Medicine, The Second Affiliated Hospital of Guilin Medical University, Guilin, 541199, ChinaThe deposition of Aβ42 has been regarded as one of the important pathological features of Alzheimer’s disease (AD). However, drug development for Aβ42 toxicity has been progressed slowly.https://doi.org/10.1515/tnsci-2022-0338alzheimer’s diseaseaβarc drosophila trehalosemitochondriamovement ability |
| spellingShingle | Li Liangxian Huang Zhiheng Wu Mingli Li Xia Xiao Bo Yao Dong Mo Biwen Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria Translational Neuroscience alzheimer’s disease aβarc drosophila trehalose mitochondria movement ability |
| title | Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria |
| title_full | Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria |
| title_fullStr | Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria |
| title_full_unstemmed | Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria |
| title_short | Trehalose improves the movement ability of Aβarc Drosophila by restoring the damaged mitochondria |
| title_sort | trehalose improves the movement ability of aβarc drosophila by restoring the damaged mitochondria |
| topic | alzheimer’s disease aβarc drosophila trehalose mitochondria movement ability |
| url | https://doi.org/10.1515/tnsci-2022-0338 |
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