IL-33 drives the antitumour effects of dendritic cells via upregulating CYLD expression in pulmonary adenocarcinoma

IL-33 drives the antitumour effects of dendritic cells via upregulating CYLD expression in pulmonary adenocarcinoma

Lung adenocarcinoma is one of the leading causes of cancer-related death worldwide. Low expression of Interleukin-33 (IL-33) was reported to be associated with the progression of pulmonary adenocarcinoma. However, the IL-33-mediated immunoregulation in pulmonary adenocarcinoma remains unclear. In th...

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Bibliographic Details
Main Authors: Jiguang Zhang, Yangming Chen, Kai Chen, Yunchao Huang, Xunyu Xu, Qianshun Chen, Chen Huang, Jiewei Luo, Xing Lin
Format: Article
Language:English
Published: Taylor & Francis Group 2019-12-01
Series:Artificial Cells, Nanomedicine, and Biotechnology
Subjects:
Interleukin 33
pulmonary adenocarcinoma
DCs
CYLD
CD4+ T cells
IL-17
Online Access:https://www.tandfonline.com/doi/10.1080/21691401.2019.1596926

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