Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation

Reactive oxygen species (ROS) derived from the phagocyte NADPH oxidase (NOX2) are essential for host defence and immunoregulation. Their levels must be tightly controlled. ROS are required to prevent infection and are used in signalling to regulate several processes that are essential for normal imm...

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Main Authors: Paige M. Mortimer, Stacey A. Mc Intyre, David C. Thomas
Format: Article
Language:English
Published: Frontiers Media S.A. 2021-09-01
Series:Frontiers in Immunology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2021.733918/full
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author Paige M. Mortimer
Stacey A. Mc Intyre
David C. Thomas
author_facet Paige M. Mortimer
Stacey A. Mc Intyre
David C. Thomas
author_sort Paige M. Mortimer
collection DOAJ
description Reactive oxygen species (ROS) derived from the phagocyte NADPH oxidase (NOX2) are essential for host defence and immunoregulation. Their levels must be tightly controlled. ROS are required to prevent infection and are used in signalling to regulate several processes that are essential for normal immunity. A lack of ROS then leads to immunodeficiency and autoinflammation. However, excess ROS are also deleterious, damaging tissues by causing oxidative stress. In this review, we focus on two particular aspects of ROS biology: (i) the emerging understanding that NOX2-derived ROS play a pivotal role in the development and maintenance of adaptive immunity and (ii) the effects of excess ROS in systemic disease and how limiting ROS might represent a therapeutic avenue in limiting excess inflammation.
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spelling doaj.art-4f32564fc2aa4ec89f6e404ed15168102022-12-21T19:11:36ZengFrontiers Media S.A.Frontiers in Immunology1664-32242021-09-011210.3389/fimmu.2021.733918733918Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and InflammationPaige M. MortimerStacey A. Mc IntyreDavid C. ThomasReactive oxygen species (ROS) derived from the phagocyte NADPH oxidase (NOX2) are essential for host defence and immunoregulation. Their levels must be tightly controlled. ROS are required to prevent infection and are used in signalling to regulate several processes that are essential for normal immunity. A lack of ROS then leads to immunodeficiency and autoinflammation. However, excess ROS are also deleterious, damaging tissues by causing oxidative stress. In this review, we focus on two particular aspects of ROS biology: (i) the emerging understanding that NOX2-derived ROS play a pivotal role in the development and maintenance of adaptive immunity and (ii) the effects of excess ROS in systemic disease and how limiting ROS might represent a therapeutic avenue in limiting excess inflammation.https://www.frontiersin.org/articles/10.3389/fimmu.2021.733918/fullNOX2ROSCGDoxidative stresssystemic inflammation
spellingShingle Paige M. Mortimer
Stacey A. Mc Intyre
David C. Thomas
Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation
Frontiers in Immunology
NOX2
ROS
CGD
oxidative stress
systemic inflammation
title Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation
title_full Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation
title_fullStr Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation
title_full_unstemmed Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation
title_short Beyond the Extra Respiration of Phagocytosis: NADPH Oxidase 2 in Adaptive Immunity and Inflammation
title_sort beyond the extra respiration of phagocytosis nadph oxidase 2 in adaptive immunity and inflammation
topic NOX2
ROS
CGD
oxidative stress
systemic inflammation
url https://www.frontiersin.org/articles/10.3389/fimmu.2021.733918/full
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