Mitochondrial function in macrophages controls cardiac repair after myocardial infarction
Cardiac healing following acute myocardial infarction (MI) involves the mobilization and activation of immune cells, including macrophages. In the early phase after MI, macrophages adopt a proinflammatory phenotype, while polarizing toward a reparative one in the late stage. Although metabolic repro...
Main Authors: | David Weissman, Christoph Maack |
---|---|
Format: | Article |
Language: | English |
Published: |
American Society for Clinical Investigation
2023-02-01
|
Series: | The Journal of Clinical Investigation |
Online Access: | https://doi.org/10.1172/JCI167079 |
Similar Items
-
Mitochondrial dysfunction in macrophages promotes inflammation and suppresses repair after myocardial infarction
by: Shanshan Cai, et al.
Published: (2023-02-01) -
Hydrogel modulate macrophages to promote myocardial repair after myocardial infarction
by: HUANG Huihui, et al.
Published: (2024-05-01) -
IKKε-deficient macrophages impede cardiac repair after myocardial infarction by enhancing the macrophage–myofibroblast transition
by: Hyang Hee Cho, et al.
Published: (2024-09-01) -
Ginsenoside Rd Promotes Cardiac Repair After Myocardial Infarction by Modulating Monocytes/Macrophages Subsets Conversion
by: Zhao T, et al.
Published: (2022-08-01) -
Cardiac macrophages and emerging roles for their metabolism after myocardial infarction
by: Edward B. Thorp
Published: (2023-09-01)