Role of Epicardial Adipose Tissue in Triggering and Maintaining Atrial Fibrillation
Atrial fibrillation is the most common arrhythmia leading to cardiogenic stroke. Without membranous sructure between epicardial adipose tissue and atrial myocardium, epicardial adipose tissue directly covers the surface of the atrial myocardium. The formation of an epicardial adipose tissue inflamma...
Main Authors: | , , , , , |
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Format: | Article |
Language: | English |
Published: |
Compuscript Ltd
2022-08-01
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Series: | Cardiovascular Innovations and Applications |
Online Access: | https://www.scienceopen.com/hosted-document?doi=10.15212/CVIA.2022.0012 |
Summary: | Atrial fibrillation is the most common arrhythmia leading to cardiogenic stroke. Without membranous sructure between epicardial adipose tissue and atrial myocardium, epicardial adipose tissue directly covers the surface of the atrial myocardium. The formation of an epicardial adipose tissue inflammatory microenvironment, fibrosis, infiltration by epicardial adipose tissue, autonomic dysfunction and oxidative stress are important mechanisms that trigger and maintain atrial fibrillation. Those mechanisms are reviewed herein. |
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ISSN: | 2009-8618 2009-8782 |