The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).

Immune semaphorins are a large family of proteins involved in the pathogenesis of inflammatory diseases through the regulation of immune homeostasis and tissue inflammation. We aim to assess the possible involvement of semaphorin3A (sema3A) and 4A (sema4A) in peripheral immune responses and bowel ti...

Full description

Bibliographic Details
Main Authors: Zahava Vadasz, Tova Rainis, Afif Nakhleh, Tharwat Haj, Jacob Bejar, Katty Halasz, Elias Toubi
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2015-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC4433250?pdf=render
_version_ 1828524148101480448
author Zahava Vadasz
Tova Rainis
Afif Nakhleh
Tharwat Haj
Jacob Bejar
Katty Halasz
Elias Toubi
author_facet Zahava Vadasz
Tova Rainis
Afif Nakhleh
Tharwat Haj
Jacob Bejar
Katty Halasz
Elias Toubi
author_sort Zahava Vadasz
collection DOAJ
description Immune semaphorins are a large family of proteins involved in the pathogenesis of inflammatory diseases through the regulation of immune homeostasis and tissue inflammation. We aim to assess the possible involvement of semaphorin3A (sema3A) and 4A (sema4A) in peripheral immune responses and bowel tissue inflammation of patients suffering from Crohn's disease (CD) and ulcerative colitis (UC).Twenty-seven CD patients and 10 UC patients were studied and compared to 10 patients followed for acute diverticulitis (disease control) and 12 healthy individuals. All were evaluated for sema3A expression on T regulatory cells (Tregs), serum levels of sema3A and sema4A, and tissue expression of sema3A and sema4A in bowel biopsies.The percentage (%) of T regulatory cells (Tregs) expressing sema3A in patients with active CD (64.5% ± 14.49%) and active UC (49.8% ± 16.45%) was significantly lower when compared to that of healthy controls (88.7% ± 3.6%, p< 0.001 and p< 0.0001, respectively). This expression was seen to be in negative correlation with CD activity. Serum levels of Sema4A were significantly lower in patients with CD and UC when compared to that of controls (5.69 ± 1 .48 ng\ml for CD, 5.26 ± 1.23 ng/ml for UC patients vs 9.74 ± 2.73 ng/ml for normal controls, P<0.001). Sema4A was highly expressed in lymphocytes of the lamina propria of CD and UC patients but absent in patients with diverticulitis or in normal individuals.Altered % of Tregs expressing sema3A in patients with inflammatory bowel diseases (IBD) is partially responsible for their failure in preventing CD4+ effector T cell induced inflammation in IBD in peripheral blood. The increased expression of sema4A in bowel biopsies from CD and UC patients is suggestive of its central role in regulating local tissue inflammation in the bowel.
first_indexed 2024-12-11T20:41:00Z
format Article
id doaj.art-783597e1e35d4dcbbea818a0fb285ff8
institution Directory Open Access Journal
issn 1932-6203
language English
last_indexed 2024-12-11T20:41:00Z
publishDate 2015-01-01
publisher Public Library of Science (PLoS)
record_format Article
series PLoS ONE
spelling doaj.art-783597e1e35d4dcbbea818a0fb285ff82022-12-22T00:51:30ZengPublic Library of Science (PLoS)PLoS ONE1932-62032015-01-01105e012586010.1371/journal.pone.0125860The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).Zahava VadaszTova RainisAfif NakhlehTharwat HajJacob BejarKatty HalaszElias ToubiImmune semaphorins are a large family of proteins involved in the pathogenesis of inflammatory diseases through the regulation of immune homeostasis and tissue inflammation. We aim to assess the possible involvement of semaphorin3A (sema3A) and 4A (sema4A) in peripheral immune responses and bowel tissue inflammation of patients suffering from Crohn's disease (CD) and ulcerative colitis (UC).Twenty-seven CD patients and 10 UC patients were studied and compared to 10 patients followed for acute diverticulitis (disease control) and 12 healthy individuals. All were evaluated for sema3A expression on T regulatory cells (Tregs), serum levels of sema3A and sema4A, and tissue expression of sema3A and sema4A in bowel biopsies.The percentage (%) of T regulatory cells (Tregs) expressing sema3A in patients with active CD (64.5% ± 14.49%) and active UC (49.8% ± 16.45%) was significantly lower when compared to that of healthy controls (88.7% ± 3.6%, p< 0.001 and p< 0.0001, respectively). This expression was seen to be in negative correlation with CD activity. Serum levels of Sema4A were significantly lower in patients with CD and UC when compared to that of controls (5.69 ± 1 .48 ng\ml for CD, 5.26 ± 1.23 ng/ml for UC patients vs 9.74 ± 2.73 ng/ml for normal controls, P<0.001). Sema4A was highly expressed in lymphocytes of the lamina propria of CD and UC patients but absent in patients with diverticulitis or in normal individuals.Altered % of Tregs expressing sema3A in patients with inflammatory bowel diseases (IBD) is partially responsible for their failure in preventing CD4+ effector T cell induced inflammation in IBD in peripheral blood. The increased expression of sema4A in bowel biopsies from CD and UC patients is suggestive of its central role in regulating local tissue inflammation in the bowel.http://europepmc.org/articles/PMC4433250?pdf=render
spellingShingle Zahava Vadasz
Tova Rainis
Afif Nakhleh
Tharwat Haj
Jacob Bejar
Katty Halasz
Elias Toubi
The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).
PLoS ONE
title The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).
title_full The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).
title_fullStr The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).
title_full_unstemmed The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).
title_short The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs).
title_sort involvement of immune semaphorins in the pathogenesis of inflammatory bowel diseases ibds
url http://europepmc.org/articles/PMC4433250?pdf=render
work_keys_str_mv AT zahavavadasz theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT tovarainis theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT afifnakhleh theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT tharwathaj theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT jacobbejar theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT kattyhalasz theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT eliastoubi theinvolvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT zahavavadasz involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT tovarainis involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT afifnakhleh involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT tharwathaj involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT jacobbejar involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT kattyhalasz involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds
AT eliastoubi involvementofimmunesemaphorinsinthepathogenesisofinflammatoryboweldiseasesibds