Anti-phospholipase A2 Receptor Antibody-Positive Membranous Glomerulopathy due to Anti-Koch’s Therapy in a Tuberculosis Patient

Tuberculosis (TB)-associated glomerulonephritis is difficult to diagnose that usually presents with hematuria, proteinuria, edema, hypertension, or renal insufficiency, which is similar to symptoms of primary glomerulonephritis. Membranous nephropathy (MN) is uncommonly seen in TB patients. We repor...

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Bibliographic Details
Main Authors: Manish R. Balwani, Amit S. Pasari, Amol Bhawane, Priyanka Tolani
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2021-01-01
Series:Saudi Journal of Kidney Diseases and Transplantation
Online Access:http://www.sjkdt.org/article.asp?issn=1319-2442;year=2021;volume=32;issue=6;spage=1800;epage=1803;aulast=Balwani
Description
Summary:Tuberculosis (TB)-associated glomerulonephritis is difficult to diagnose that usually presents with hematuria, proteinuria, edema, hypertension, or renal insufficiency, which is similar to symptoms of primary glomerulonephritis. Membranous nephropathy (MN) is uncommonly seen in TB patients. We report a case of a 30-year-old female with Koch’s chest who developed anti-phospholipase A2 receptor antibody-positive MN after initiation of anti-Koch’s therapy and resolved after completion of anti-Koch’s therapy.
ISSN:1319-2442