RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING

<p>The problem of vascular aging is discussed. Special attention among the signs of vascular aging is paid to the activation of the renin-angiotensin-aldosterone system as a source of chronic inflammation and oxidative stress, as well as to its relation to replicative cellular senescence. Pote...

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Main Authors: V. S. Pykhtina, I. D. Strazhesko, M. V. Agaltsov, O. N. Tkacheva
Format: Article
Language:English
Published: Столичная издательская компания 2015-09-01
Series:Рациональная фармакотерапия в кардиологии
Subjects:
Online Access:http://www.rpcardio.ru/jour/article/view/257
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author V. S. Pykhtina
I. D. Strazhesko
M. V. Agaltsov
O. N. Tkacheva
author_facet V. S. Pykhtina
I. D. Strazhesko
M. V. Agaltsov
O. N. Tkacheva
author_sort V. S. Pykhtina
collection DOAJ
description <p>The problem of vascular aging is discussed. Special attention among the signs of vascular aging is paid to the activation of the renin-angiotensin-aldosterone system as a source of chronic inflammation and oxidative stress, as well as to its relation to replicative cellular senescence. Potential routes of exposure to these processes are also considered.</p>
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publishDate 2015-09-01
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series Рациональная фармакотерапия в кардиологии
spelling doaj.art-82450bf0afe547c882543a2c117bd4ef2023-09-02T07:22:05ZengСтоличная издательская компанияРациональная фармакотерапия в кардиологии1819-64462225-36532015-09-0110331231610.1234/1819-6446-2014-3-312-316257RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEINGV. S. Pykhtina0I. D. Strazhesko1M. V. Agaltsov2O. N. Tkacheva3Государственный научно-исследовательский центр профилактической медициныГосударственный научно-исследовательский центр профилактической медициныГосударственный научно-исследовательский центр профилактической медициныГосударственный научно-исследовательский центр профилактической медицины<p>The problem of vascular aging is discussed. Special attention among the signs of vascular aging is paid to the activation of the renin-angiotensin-aldosterone system as a source of chronic inflammation and oxidative stress, as well as to its relation to replicative cellular senescence. Potential routes of exposure to these processes are also considered.</p>http://www.rpcardio.ru/jour/article/view/257сосудистое старениеренин-ангиотензин-альдостероновая системарепликативное клеточное старение
spellingShingle V. S. Pykhtina
I. D. Strazhesko
M. V. Agaltsov
O. N. Tkacheva
RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING
Рациональная фармакотерапия в кардиологии
сосудистое старение
ренин-ангиотензин-альдостероновая система
репликативное клеточное старение
title RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING
title_full RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING
title_fullStr RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING
title_full_unstemmed RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING
title_short RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM AND REPLICATIVE CELLULAR SENESCENCE: THEIR INTERACTION DURING THE VASCULAR AGEING
title_sort renin angiotensin aldosterone system and replicative cellular senescence their interaction during the vascular ageing
topic сосудистое старение
ренин-ангиотензин-альдостероновая система
репликативное клеточное старение
url http://www.rpcardio.ru/jour/article/view/257
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AT idstrazhesko reninangiotensinaldosteronesystemandreplicativecellularsenescencetheirinteractionduringthevascularageing
AT mvagaltsov reninangiotensinaldosteronesystemandreplicativecellularsenescencetheirinteractionduringthevascularageing
AT ontkacheva reninangiotensinaldosteronesystemandreplicativecellularsenescencetheirinteractionduringthevascularageing