Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
Alzheimer’s disease (AD) is a neurodegeneration disease. The previous work from our research group demonstrated the neuroprotective effects of tripchlorolide (T4) in AD animal models.
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Format: | Article |
Language: | English |
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De Gruyter
2021-07-01
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Series: | Translational Neuroscience |
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Online Access: | https://doi.org/10.1515/tnsci-2020-0178 |
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author | Zeng Yuqi Li Yongkun Shen Hui Lin Nan Zhang Jian |
author_facet | Zeng Yuqi Li Yongkun Shen Hui Lin Nan Zhang Jian |
author_sort | Zeng Yuqi |
collection | DOAJ |
description | Alzheimer’s disease (AD) is a neurodegeneration disease. The previous work from our research group demonstrated the neuroprotective effects of tripchlorolide (T4) in AD animal models. |
first_indexed | 2024-04-13T01:17:51Z |
format | Article |
id | doaj.art-8312c712d0b342719baeba14c39c5b8e |
institution | Directory Open Access Journal |
issn | 2081-6936 |
language | English |
last_indexed | 2024-04-13T01:17:51Z |
publishDate | 2021-07-01 |
publisher | De Gruyter |
record_format | Article |
series | Translational Neuroscience |
spelling | doaj.art-8312c712d0b342719baeba14c39c5b8e2022-12-22T03:08:52ZengDe GruyterTranslational Neuroscience2081-69362021-07-0112130130810.1515/tnsci-2020-0178Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cellsZeng Yuqi0Li Yongkun1Shen Hui2Lin Nan3Zhang Jian4Department of Neurology, Institute of Clinical Neurology, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaDepartment of Neurology, Xiamen Key Laboratory of Brain Center, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian, 361002, ChinaDepartment of Geriatrics, Fujian Institute of Geriatrics, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaDepartment of Geriatrics, Fujian Institute of Geriatrics, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaDepartment of Neurology, Institute of Clinical Neurology, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaAlzheimer’s disease (AD) is a neurodegeneration disease. The previous work from our research group demonstrated the neuroprotective effects of tripchlorolide (T4) in AD animal models.https://doi.org/10.1515/tnsci-2020-0178tripchlorolideneprilysinamyloid-β protein, n2a/app695 cells |
spellingShingle | Zeng Yuqi Li Yongkun Shen Hui Lin Nan Zhang Jian Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells Translational Neuroscience tripchlorolide neprilysin amyloid-β protein, n2a/app695 cells |
title | Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells |
title_full | Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells |
title_fullStr | Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells |
title_full_unstemmed | Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells |
title_short | Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells |
title_sort | tripchlorolide attenuates β amyloid generation by inducing nep activity in n2a app695 cells |
topic | tripchlorolide neprilysin amyloid-β protein, n2a/app695 cells |
url | https://doi.org/10.1515/tnsci-2020-0178 |
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