Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells

Alzheimer’s disease (AD) is a neurodegeneration disease. The previous work from our research group demonstrated the neuroprotective effects of tripchlorolide (T4) in AD animal models.

Bibliographic Details
Main Authors: Zeng Yuqi, Li Yongkun, Shen Hui, Lin Nan, Zhang Jian
Format: Article
Language:English
Published: De Gruyter 2021-07-01
Series:Translational Neuroscience
Subjects:
Online Access:https://doi.org/10.1515/tnsci-2020-0178
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author Zeng Yuqi
Li Yongkun
Shen Hui
Lin Nan
Zhang Jian
author_facet Zeng Yuqi
Li Yongkun
Shen Hui
Lin Nan
Zhang Jian
author_sort Zeng Yuqi
collection DOAJ
description Alzheimer’s disease (AD) is a neurodegeneration disease. The previous work from our research group demonstrated the neuroprotective effects of tripchlorolide (T4) in AD animal models.
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issn 2081-6936
language English
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spelling doaj.art-8312c712d0b342719baeba14c39c5b8e2022-12-22T03:08:52ZengDe GruyterTranslational Neuroscience2081-69362021-07-0112130130810.1515/tnsci-2020-0178Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cellsZeng Yuqi0Li Yongkun1Shen Hui2Lin Nan3Zhang Jian4Department of Neurology, Institute of Clinical Neurology, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaDepartment of Neurology, Xiamen Key Laboratory of Brain Center, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian, 361002, ChinaDepartment of Geriatrics, Fujian Institute of Geriatrics, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaDepartment of Geriatrics, Fujian Institute of Geriatrics, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaDepartment of Neurology, Institute of Clinical Neurology, Fujian Medical University Union Hospital, 29 Xinquan Road, Fuzhou, Fujian, 350001, ChinaAlzheimer’s disease (AD) is a neurodegeneration disease. The previous work from our research group demonstrated the neuroprotective effects of tripchlorolide (T4) in AD animal models.https://doi.org/10.1515/tnsci-2020-0178tripchlorolideneprilysinamyloid-β protein, n2a/app695 cells
spellingShingle Zeng Yuqi
Li Yongkun
Shen Hui
Lin Nan
Zhang Jian
Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
Translational Neuroscience
tripchlorolide
neprilysin
amyloid-β protein, n2a/app695 cells
title Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
title_full Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
title_fullStr Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
title_full_unstemmed Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
title_short Tripchlorolide attenuates β-amyloid generation by inducing NEP activity in N2a/APP695 cells
title_sort tripchlorolide attenuates β amyloid generation by inducing nep activity in n2a app695 cells
topic tripchlorolide
neprilysin
amyloid-β protein, n2a/app695 cells
url https://doi.org/10.1515/tnsci-2020-0178
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AT liyongkun tripchlorolideattenuatesbamyloidgenerationbyinducingnepactivityinn2aapp695cells
AT shenhui tripchlorolideattenuatesbamyloidgenerationbyinducingnepactivityinn2aapp695cells
AT linnan tripchlorolideattenuatesbamyloidgenerationbyinducingnepactivityinn2aapp695cells
AT zhangjian tripchlorolideattenuatesbamyloidgenerationbyinducingnepactivityinn2aapp695cells