Author Correction: Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation

Author Correction: Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation

Bibliographic Details
Main Authors:	Julia Schewe, Eric Seidel, Sofia Forslund, Lajos Marko, Jörg Peters, Dominik N. Muller, Christoph Fahlke, Gabriel Stölting, Ute Scholl
Format:	Article
Language:	English
Published:	Nature Portfolio 2022-05-01
Series:	Nature Communications
Online Access:	https://doi.org/10.1038/s41467-022-29242-3

Similar Items

CLC channel function and dysfunction in health and disease
by: Gabriel eStölting, et al.
Published: (2014-10-01)

Diagnostic performance of CT versus MR in detecting aldosterone-producing adenoma in primary hyperaldosteronism (Conn's syndrome).
by: Lingam, R, et al.
Published: (2004)

Redefining primary hyperaldosteronism as “The Syndrome of Inappropriate Aldosterone Secretion (SIALDS)”: A common but unrecognized cause of hypertension
by: Rajat Lamba
Published: (2023-12-01)

Familial Hyperaldosteronism Type 3 with a Rapidly Growing Adrenal Tumor: An In Situ Aldosterone Imaging Study
by: Nae Takizawa, et al.
Published: (2021-12-01)

Structure of the human ClC-1 chloride channel.
by: Kaituo Wang, et al.
Published: (2019-04-01)

ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway
by: Juan Sierra-Marquez, et al.
Published: (2022-08-01)

The dimerization equilibrium of a ClC Cl−/H+ antiporter in lipid bilayers
by: Rahul Chadda, et al.
Published: (2016-08-01)

CUL4-DDB1-CRBN E3 Ubiquitin Ligase Regulates Proteostasis of ClC-2 Chloride Channels: Implication for Aldosteronism and Leukodystrophy
by: Ssu-Ju Fu, et al.
Published: (2020-05-01)

Corrigendum: ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway
by: Juan Sierra-Marquez, et al.
Published: (2023-04-01)

New insights into the mechanism of NO3- selectivity in the human kidney chloride channel ClC-Ka and the CLC protein family
by: Lagostena, L, et al.
Published: (2019)

Backbone amides are determinants of Cl− selectivity in CLC ion channels
by: Lilia Leisle, et al.
Published: (2022-12-01)

Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalities
by: Gabriel Stölting, et al.
Published: (2023-10-01)

Primary idiopathic hyperaldosteronism in clinical practice
Published: (2012-12-01)

A CLC-ec1 mutant reveals global conformational change and suggests a unifying mechanism for the CLC Cl–/H+ transport cycle
by: Tanmay S Chavan, et al.
Published: (2020-04-01)

The Role of the Lysosomal Cl<sup>−</sup>/H<sup>+</sup> Antiporter ClC-7 in Osteopetrosis and Neurodegeneration
by: Giovanni Zifarelli
Published: (2022-01-01)

Genetic Ablation of the ClC-2 Cl- Channel Disrupts Mouse Gastric Parietal Cell Acid Secretion.
by: Meghali P Nighot, et al.
Published: (2015-01-01)

In the Refractory Hypertension “Labyrinth”. Focus on Primary Hyperaldosteronism
by: O. V. Tsygankova, et al.
Published: (2020-09-01)

Pathological classification and surgical prognosis of idiopathic hyperaldosteronism
by: WANG Hui-ping, CUI Yun-ying, MA Xiao-sen, WEN Jin, REN Wei-dong, TONG An-li
Published: (2021-10-01)

Comparative ontogeny, processing, and segmental distribution of the renal chloride channel, ClC-5.
by: Jouret, F, et al.
Published: (2004)

Divergent Cl- and H+ pathways underlie transport coupling and gating in CLC exchangers and channels
by: Lilia Leisle, et al.
Published: (2020-04-01)

The CLC-5 2Cl-/H+ exchange transporter in endosomal function and Dent’s disease.
by: Jonathan eLippiat, et al.
Published: (2012-11-01)

<i>Modus operandi</i> of ClC-K2 Cl<sup>−</sup> Channel in the Collecting Duct Intercalated Cells
by: Anna Stavniichuk, et al.
Published: (2023-01-01)

Epigenetic and transcriptomic alterations in the ClC-3-deficient mice consuming a normal diet
by: Zhenghui Jing, et al.
Published: (2023-05-01)

Revealing an outward-facing open conformational state in a CLC Cl–/H+ exchange transporter
by: Chandra M Khantwal, et al.
Published: (2016-01-01)

Primary hyperaldosteronism (Conn syndrome): MR imaging findings.
by: Sohaib, SA, et al.
Published: (2000)

The problem of timely diagnosis of primary hyperaldosteronism. Clinical case
by: V.M. Zhdan, et al.
Published: (2023-10-01)

Mutational analysis of CLC-5, cofilin and CLC-4 in patients with Dent's disease.
by: Wu, F, et al.
Published: (2009)

Association of Elevated Serum Aldosterone Concentrations in Pregnancy with Hypertension
by: Robin Shoemaker, et al.
Published: (2023-11-01)

CLC anion/proton exchangers regulate secretory vesicle filling and granule exocytosis in chromaffin cells
by: Comini, M, et al.
Published: (2022)

Deep Westerbork 1.4 GH[CLC]z[/CLC] Imaging of the Bootes Field
by: de Vries, W, et al.
Published: (2002)

The role of the endosomal chloride/proton antiporter ClC-5 in proximal tubule endocytosis and kidney physiology
by: Comini, M, et al.
Published: (2021)

ClC-1 Chloride Channel: Inputs on the Structure–Function Relationship of Myotonia Congenita-Causing Mutations
by: Oscar Brenes, et al.
Published: (2023-09-01)

Regulation of ClC-2 Chloride Channel Proteostasis by Molecular Chaperones: Correction of Leukodystrophy-Associated Defect
by: Ssu-Ju Fu, et al.
Published: (2021-05-01)

Chaperone activity of niflumic acid on ClC-1 chloride channel mutants causing myotonia congenita
by: Concetta Altamura, et al.
Published: (2022-08-01)

Primary Hyperaldosteronism: When to Suspect It and How to Confirm Its Diagnosis
by: Jorge Gabriel Ruiz-Sánchez, et al.
Published: (2022-01-01)

A hyperaldosteronism subtypes predictive model using ensemble learning
by: Shigehiro Karashima, et al.
Published: (2023-02-01)

Screening of Hyperaldosteronism on the Investigation of Secondary Hypertension: Single-centre Experience
by: Gülsüm Bingöl, et al.
Published: (2023-02-01)

Impaired Autophagic Clearance with a Gain-of-Function Variant of the Lysosomal Cl<sup>−</sup>/H<sup>+</sup> Exchanger ClC-7
by: Shroddha Bose, et al.
Published: (2023-12-01)

Regulated insulin release requires chloride channel 3 (ClC3) for proper granule acidification and priming
by: Li, D, et al.
Published: (2005)

The ClC-5 knockout mouse model of Dent's disease has renal hypercalciuria and increased bone turnover.
by: Silva, I, et al.
Published: (2003)