Differential ER stress as a driver of cell fate following ricin toxin exposure

Differential ER stress as a driver of cell fate following ricin toxin exposure

Abstract Inhalation of trace amounts of ricin toxin, a plant‐derived ribosome‐inactivating protein, results in ablation of alveolar macrophages, widespread epithelial damage, and the onset of acute respiratory distress syndrome (ARDS). While ricin's receptors are ubiquitous, certain cell types...

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Bibliographic Details
Main Authors: Claire Peterson‐Reynolds, Nicholas J. Mantis
Format: Article
Language:English
Published: Wiley 2022-01-01
Series:FASEB BioAdvances
Subjects:
apoptosis
epithelium
inflammation
lung
macrophage
stress
Online Access:https://doi.org/10.1096/fba.2021-00005

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