Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress

Bibliographic Details
Main Authors: Meiying Yang, Jie Sun, David F. Stowe, Emad Tajkhorshid, Wai-Meng Kwok, Amadou K. S. Camara
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2020-09-01
Series:Cellular Physiology and Biochemistry
Online Access:https://www.cellphysiolbiochem.com/Articles/000274/
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author Meiying Yang
Jie Sun
David F. Stowe
Emad Tajkhorshid
Wai-Meng Kwok
Amadou K. S. Camara
author_facet Meiying Yang
Jie Sun
David F. Stowe
Emad Tajkhorshid
Wai-Meng Kwok
Amadou K. S. Camara
author_sort Meiying Yang
collection DOAJ
first_indexed 2024-12-11T03:26:06Z
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institution Directory Open Access Journal
issn 1015-8987
language English
last_indexed 2024-12-11T03:26:06Z
publishDate 2020-09-01
publisher Cell Physiol Biochem Press GmbH & Co KG
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spelling doaj.art-8b759b52b15a48f99ef5c8919dfe77ea2022-12-22T01:22:30ZengCell Physiol Biochem Press GmbH & Co KGCellular Physiology and Biochemistry1015-89872020-09-0154585387410.33594/000000274Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic StressMeiying Yang0Jie Sun1 David F. Stowe2Emad Tajkhorshid3Wai-Meng Kwok4Amadou K. S. Camara5Department of Anesthesiology, Medical College of Wisconsin, Milwaukee, WI, USADepartment of Anesthesiology, Medical College of Wisconsin, Milwaukee, WI, USA; Institute of Clinical Medicine Research, Department of Gastroenterology and Hepatology, Suzhou Hospital affiliated with Nanjing Medical University, Suzhou, Jiangsu, ChinaDepartment of Anesthesiology, Medical College of Wisconsin, Milwaukee, WI, USA; Research Service, Zablocki VA Medical Center, Milwaukee, WI, USA; Department of Physiology, Medical College of Wisconsin, Milwaukee, WI, USA; Cardiovascular Center, Medical College of Wisconsin, Milwaukee, WI, USANIH Center for Macromolecular Modeling and Bioinformatics, Beckman Institute for Advanced Science and Technology, University of Illinois at Urbana Champaign, Urbana, IL, USA; Department of Biochemistry, and Center for Biophysics and Quantitative Biology, University of Illinois at Urbana Champaign, Urbana, IL, USADepartment of Anesthesiology, Medical College of Wisconsin, Milwaukee, WI, USA; Cardiovascular Center, Medical College of Wisconsin, Milwaukee, WI, USA; Cancer Center, Medical College of Wisconsin, Milwaukee, WI, USA; Department of Pharmacology and Toxicology, Medical College of Wisconsin, Milwaukee, WI, USADepartment of Anesthesiology, Medical College of Wisconsin, Milwaukee, WI, USA; Department of Physiology, Medical College of Wisconsin, Milwaukee, WI, USA; Cardiovascular Center, Medical College of Wisconsin, Milwaukee, WI, USA; Cancer Center, Medical College of Wisconsin, Milwaukee, WI, USAhttps://www.cellphysiolbiochem.com/Articles/000274/
spellingShingle Meiying Yang
Jie Sun
David F. Stowe
Emad Tajkhorshid
Wai-Meng Kwok
Amadou K. S. Camara
Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress
Cellular Physiology and Biochemistry
title Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress
title_full Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress
title_fullStr Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress
title_full_unstemmed Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress
title_short Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress
title_sort knockout of vdac1 in h9c2 cells promotes oxidative stress induced cell apoptosis through decreased mitochondrial hexokinase ii binding and enhanced glycolytic stress
url https://www.cellphysiolbiochem.com/Articles/000274/
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