Early detachment of neuromuscular junction proteins in ALS mice with SODG93A mutation

Early detachment of neuromuscular junction proteins in ALS mice with SODG93A mutation

The transgenic animals with mutant copper/zinc superoxide dismutase (SOD1) DNA develop paralytic motor neuron disease resembling human amyotrophic lateral sclerosis (ALS) patients and are commonly used as models for ALS. In the transgenic (Tg) mice with the G93A mutation of the human SOD1 gene (SOD1...

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Bibliographic Details
Main Authors: Hisashi Narai, Yasuhiro Manabe, Makiko Nagai, Isao Nagano, Yasuyuki Ohta, Tetsuro Murakami, Yasushi Takehisa, Tatsushi Kamiya, Koji Abe
Format: Article
Language:English
Published: MDPI AG 2009-11-01
Series:Neurology International
Subjects:
amyotrophic lateral sclerosis
neuromuscular junction
α-bungarotoxin
SV-2
agrin
Online Access:https://www.pagepress.org/journals/index.php/ni/article/view/221

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