The Potential for Placental Activation of PPARγ to Improve the Angiogenic Profile in Preeclampsia
Preeclampsia (PE) is one of the most common causes of maternal-fetal morbidity and mortality world-wide. While the underlying causes of PE remain elusive, aberrant trophoblast differentiation and function are thought to cause an imbalance of secreted angiogenic proteins resulting in systemic endothe...
Main Authors: | Brooke Grimaldi, Hamid-Reza Kohan-Ghadr, Sascha Drewlo |
---|---|
Format: | Article |
Language: | English |
Published: |
MDPI AG
2022-11-01
|
Series: | Cells |
Subjects: | |
Online Access: | https://www.mdpi.com/2073-4409/11/21/3514 |
Similar Items
-
PPARγ Regulates Triclosan Induced Placental Dysfunction
by: Jing Li, et al.
Published: (2021-12-01) -
Understanding PPARγ and Its Agonists on Trophoblast Differentiation and Invasion: Potential Therapeutic Targets for Gestational Diabetes Mellitus and Preeclampsia
by: Yushu Qin, et al.
Published: (2023-05-01) -
C1431T Variant of PPARγ Is Associated with Preeclampsia in Pregnant Women
by: Fulin Liu, et al.
Published: (2021-10-01) -
PPARγ—A Factor Linking Metabolically Unhealthy Obesity with Placental Pathologies
by: Sebastian Kwiatkowski, et al.
Published: (2021-12-01) -
Irisin Protects the Human Placenta from Oxidative Stress and Apoptosis via Activation of the Akt Signaling Pathway
by: Hamid-Reza Kohan-Ghadr, et al.
Published: (2021-10-01)