LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE.
Main Authors: | , , , |
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Format: | Article |
Language: | English |
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Elsevier
2023-10-01
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Series: | IBRO Neuroscience Reports |
Online Access: | http://www.sciencedirect.com/science/article/pii/S2667242123009855 |
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author | Laura Perez Abshana Miguel Mendivil-Perez Marlene Jiménez Del Rio Carlos Vélez Pardo |
author_facet | Laura Perez Abshana Miguel Mendivil-Perez Marlene Jiménez Del Rio Carlos Vélez Pardo |
author_sort | Laura Perez Abshana |
collection | DOAJ |
first_indexed | 2024-03-11T12:32:48Z |
format | Article |
id | doaj.art-9cb9b89a3a0f42a9aca8d9f0b75eeb1b |
institution | Directory Open Access Journal |
issn | 2667-2421 |
language | English |
last_indexed | 2024-03-11T12:32:48Z |
publishDate | 2023-10-01 |
publisher | Elsevier |
record_format | Article |
series | IBRO Neuroscience Reports |
spelling | doaj.art-9cb9b89a3a0f42a9aca8d9f0b75eeb1b2023-11-06T04:24:25ZengElsevierIBRO Neuroscience Reports2667-24212023-10-0115S474LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE.Laura Perez Abshana0Miguel Mendivil-Perez1Marlene Jiménez Del Rio2Carlos Vélez Pardo3University of Antioquia (UdeA), Faculty Of Medicine, Medellin, ColombiaUniversidad De Antioquia, Neuroscience Group Of Antioquia, Medical Research Institute, Faculty Of Medicine, Medellin, ColombiaUniversidad De Antioquia, Neuroscience Group Of Antioquia, Medical Research Institute, Faculty Of Medicine, Medellin, ColombiaUniversity of Antioquia, Neuroscience Research Group, Medellín, Colombiahttp://www.sciencedirect.com/science/article/pii/S2667242123009855 |
spellingShingle | Laura Perez Abshana Miguel Mendivil-Perez Marlene Jiménez Del Rio Carlos Vélez Pardo LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE. IBRO Neuroscience Reports |
title | LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE. |
title_full | LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE. |
title_fullStr | LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE. |
title_full_unstemmed | LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE. |
title_short | LRRK2 REGULATES CELL DEATH PROGRESSION RATHER THAN AUTOPHAGIC FLUX IN AN IN VITRO MODEL OF PARKINSON’S DISEASE INDUCED BY CHEMICAL INHIBITION OF GLUCOCEREBROSIDASE. |
title_sort | lrrk2 regulates cell death progression rather than autophagic flux in an in vitro model of parkinson s disease induced by chemical inhibition of glucocerebrosidase |
url | http://www.sciencedirect.com/science/article/pii/S2667242123009855 |
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