Overexpression of CERKL Protects Retinal Pigment Epithelium Mitochondria from Oxidative Stress Effects

Overexpression of CERKL Protects Retinal Pigment Epithelium Mitochondria from Oxidative Stress Effects

The precise function of <i>CERKL</i>, a Retinitis Pigmentosa (RP) causative gene, is not yet fully understood. There is evidence that <i>CERKL</i> is involved in the regulation of autophagy, stress granules, and mitochondrial metabolism, and it is considered a gene that is re...

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Bibliographic Details
Main Authors: Rocío García-Arroyo, Aleix Gavaldà-Navarro, Francesc Villarroya, Gemma Marfany, Serena Mirra
Format: Article
Language:English
Published: MDPI AG 2021-12-01
Series:Antioxidants
Subjects:
retinitis pigmentosa
<i>CERKL</i>
retinal pigment epithelium
mitochondrial network
oxidative stress
Online Access:https://www.mdpi.com/2076-3921/10/12/2018

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