S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS

Bibliographic Details
Main Authors: Z. Shi, J. Liu, Y. Zhao, L. Yang, Y. Cai, P. Zhang, Z. Xu, T. Qin, S. Qu, L. Pan, J. Wu, X. Yan, Z. Li, W. Zhang, Y. Yan, H. Huang, G. Huang, B. Li, X. Wu, Z. Xiao
Format: Article
Language:English
Published: Wiley 2022-06-01
Series:HemaSphere
Online Access:http://journals.lww.com/10.1097/01.HS9.0000843652.05474.21
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author Z. Shi
J. Liu
Y. Zhao
L. Yang
Y. Cai
P. Zhang
Z. Xu
T. Qin
S. Qu
L. Pan
J. Wu
X. Yan
Z. Li
W. Zhang
Y. Yan
H. Huang
G. Huang
B. Li
X. Wu
Z. Xiao
author_facet Z. Shi
J. Liu
Y. Zhao
L. Yang
Y. Cai
P. Zhang
Z. Xu
T. Qin
S. Qu
L. Pan
J. Wu
X. Yan
Z. Li
W. Zhang
Y. Yan
H. Huang
G. Huang
B. Li
X. Wu
Z. Xiao
author_sort Z. Shi
collection DOAJ
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institution Directory Open Access Journal
issn 2572-9241
language English
last_indexed 2024-03-07T17:02:16Z
publishDate 2022-06-01
publisher Wiley
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spelling doaj.art-c57b97b19ce4458dabddb568d559b6a32024-03-03T03:19:20ZengWileyHemaSphere2572-92412022-06-016919210.1097/01.HS9.0000843652.05474.21202206003-00091S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMSZ. Shi0J. Liu1Y. Zhao2L. Yang3Y. Cai4P. Zhang5Z. Xu6T. Qin7S. Qu8L. Pan9J. Wu10X. Yan11Z. Li12W. Zhang13Y. Yan14H. Huang15G. Huang16B. Li17X. Wu18Z. Xiao191 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin3 Department of Cell Biology, Tianjin Medical University1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin4 pathology center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin, China1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin3 Department of Cell Biology, Tianjin Medical University1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjin5 Divisions of Pathology and Experimental Hematology and Cancer Biology, Cincinnati Children’s Hospital Medical Center, Cincinnati, United States of America6 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College7 Department of Cell Biology, Tianjin Medical University1 MDS/MPN center, State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Tianjinhttp://journals.lww.com/10.1097/01.HS9.0000843652.05474.21
spellingShingle Z. Shi
J. Liu
Y. Zhao
L. Yang
Y. Cai
P. Zhang
Z. Xu
T. Qin
S. Qu
L. Pan
J. Wu
X. Yan
Z. Li
W. Zhang
Y. Yan
H. Huang
G. Huang
B. Li
X. Wu
Z. Xiao
S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS
HemaSphere
title S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS
title_full S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS
title_fullStr S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS
title_full_unstemmed S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS
title_short S190: ASXL1 MUTATIONS ACCELERATE BONE MARROW FIBROSIS VIA EGR1-TNFA AXIS MEDIATED INFLAMMATION AND FIBROCYTE GENERATION IN MYELOPROLIFERATIVE NEOPLASMS
title_sort s190 asxl1 mutations accelerate bone marrow fibrosis via egr1 tnfa axis mediated inflammation and fibrocyte generation in myeloproliferative neoplasms
url http://journals.lww.com/10.1097/01.HS9.0000843652.05474.21
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