Copper-Zinc Superoxide Dismutase (SOD1) Is Released by Microglial Cells and Confers Neuroprotection against 6-OHDA Neurotoxicity

Copper-Zinc Superoxide Dismutase (SOD1) Is Released by Microglial Cells and Confers Neuroprotection against 6-OHDA Neurotoxicity

Microglial-neuronal interactions are essential for brain physiopathology. In this framework, recent data have changed the concept of microglia from essentially macrophagic cells to crucial elements in maintaining neuronal homeostasis and function through the release of neuroprotective molecules. Usi...

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Bibliographic Details
Main Authors: Elisabetta Polazzi, Ilaria Mengoni, Marco Caprini, Emiliano Peña-Altamira, Ewelina Kurtys, Barbara Monti
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2012-05-01
Series:Neurosignals
Subjects:
Conditioned medium
SOD1
Neuroprotection
Cerebellar granule neurons
6-Hydroxydopamine
Microglia
Online Access:http://www.karger.com/Article/FullText/337115

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