RUNX2 prompts triple negative breast cancer drug resistance through TGF-β pathway regulating breast cancer stem cells
Triple-negative breast cancer (TNBC) stands out as the most aggressive subtype within the spectrum of breast cancer. The current clinical guidelines propose treatment strategies involving cytotoxic agents like epirubicin or paclitaxel. However, the emergence of acquired resistance frequently precipi...
Κύριοι συγγραφείς: | Fengxu Lv, Wentao Si, Xiaodan Xu, Xiaogang He, Ying Wang, Yetian Li, Feifei Li |
---|---|
Μορφή: | Άρθρο |
Γλώσσα: | English |
Έκδοση: |
Elsevier
2024-02-01
|
Σειρά: | Neoplasia: An International Journal for Oncology Research |
Θέματα: | |
Διαθέσιμο Online: | http://www.sciencedirect.com/science/article/pii/S1476558624000046 |
Παρόμοια τεκμήρια
Παρόμοια τεκμήρια
-
RUNX2 facilitates aggressiveness and chemoresistance of triple negative breast cancer cells via activating MMP1
ανά: Wentao Si, κ.ά.
Έκδοση: (2022-11-01) -
RUNX1 Is Regulated by Androgen Receptor to Promote Cancer Stem Markers and Chemotherapy Resistance in Triple Negative Breast Cancer
ανά: Natalia B. Fernández, κ.ά.
Έκδοση: (2023-01-01) -
Profiling molecular regulators of recurrence in chemorefractory triple-negative breast cancers
ανά: Bradley A. Hancock, κ.ά.
Έκδοση: (2019-08-01) -
Comprehensive Analysis of RUNX and TGF-β Mediated Regulation of Immune Cell Infiltration in Breast Cancer
ανά: Liang Gao, κ.ά.
Έκδοση: (2021-08-01) -
TGF-β induced reprogramming and drug resistance in triple-negative breast cells
ανά: Guoyu Wu, κ.ά.
Έκδοση: (2022-04-01)