Is Mitochondrial Oxidative Stress a Viable Therapeutic Target in Preeclampsia?
Despite considerable research efforts over the past few decades, the pathology of preeclampsia (PE) remains poorly understood with no new FDA-approved treatments. There is a substantial amount of work being conducted by investigators around the world to identify targets to develop therapies for PE....
Main Authors: | Ramana Vaka, Evangeline Deer, Babbette LaMarca |
---|---|
Format: | Article |
Language: | English |
Published: |
MDPI AG
2022-01-01
|
Series: | Antioxidants |
Subjects: | |
Online Access: | https://www.mdpi.com/2076-3921/11/2/210 |
Similar Items
-
Characterization of Mitochondrial Bioenergetics in Preeclampsia
by: Ramana Vaka, et al.
Published: (2021-10-01) -
Tumor necrosis factor alpha (TNF-α) blockade improves natural killer cell (NK) activation, hypertension, and mitochondrial oxidative stress in a preclinical rat model of preeclampsia
by: Mark W. Cunningham, et al.
Published: (2020-10-01) -
Setting a stage: Inflammation during preeclampsia and postpartum
by: Owen Herrock, et al.
Published: (2023-02-01) -
Low Dose of IL-2 Normalizes Hypertension and Mitochondrial Function in the RUPP Rat Model of Placental Ischemia
by: Evangeline Deer, et al.
Published: (2021-10-01) -
Progesterone Induced Blocking Factor Reduces Hypertension and Placental Mitochondrial Dysfunction in Response to sFlt-1 during Pregnancy
by: Evangeline Deer, et al.
Published: (2021-10-01)