Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases
The role of mammary gland tissue-resident macrophages (MGTRMs) in the development of TNBC breast tumors and recurrence after surgery and chemotherapy is investigated.
| Main Authors: | , , , , , , , , |
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| Format: | Article |
| Language: | English |
| Published: |
Nature Portfolio
2023-02-01
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| Series: | Communications Biology |
| Online Access: | https://doi.org/10.1038/s42003-023-04525-7 |
| _version_ | 1811171591942307840 |
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| author | Ryuichiro Hirano Koki Okamoto Miyu Shinke Marika Sato Shigeaki Watanabe Hitomi Watanabe Gen Kondoh Tetsuya Kadonosono Shinae Kizaka-Kondoh |
| author_facet | Ryuichiro Hirano Koki Okamoto Miyu Shinke Marika Sato Shigeaki Watanabe Hitomi Watanabe Gen Kondoh Tetsuya Kadonosono Shinae Kizaka-Kondoh |
| author_sort | Ryuichiro Hirano |
| collection | DOAJ |
| description | The role of mammary gland tissue-resident macrophages (MGTRMs) in the development of TNBC breast tumors and recurrence after surgery and chemotherapy is investigated. |
| first_indexed | 2024-04-10T17:17:41Z |
| format | Article |
| id | doaj.art-d54fb73de5ad471e8c6fde0df3484d37 |
| institution | Directory Open Access Journal |
| issn | 2399-3642 |
| language | English |
| last_indexed | 2024-04-10T17:17:41Z |
| publishDate | 2023-02-01 |
| publisher | Nature Portfolio |
| record_format | Article |
| series | Communications Biology |
| spelling | doaj.art-d54fb73de5ad471e8c6fde0df3484d372023-02-05T12:21:02ZengNature PortfolioCommunications Biology2399-36422023-02-016111110.1038/s42003-023-04525-7Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastasesRyuichiro Hirano0Koki Okamoto1Miyu Shinke2Marika Sato3Shigeaki Watanabe4Hitomi Watanabe5Gen Kondoh6Tetsuya Kadonosono7Shinae Kizaka-Kondoh8School of Life Science and Technology, Tokyo Institute of TechnologySchool of Life Science and Technology, Tokyo Institute of TechnologySchool of Life Science and Technology, Tokyo Institute of TechnologySchool of Life Science and Technology, Tokyo Institute of TechnologySchool of Life Science and Technology, Tokyo Institute of TechnologyInstitute for Life and Medical Sciences, Kyoto University, SakyoInstitute for Life and Medical Sciences, Kyoto University, SakyoSchool of Life Science and Technology, Tokyo Institute of TechnologySchool of Life Science and Technology, Tokyo Institute of TechnologyThe role of mammary gland tissue-resident macrophages (MGTRMs) in the development of TNBC breast tumors and recurrence after surgery and chemotherapy is investigated.https://doi.org/10.1038/s42003-023-04525-7 |
| spellingShingle | Ryuichiro Hirano Koki Okamoto Miyu Shinke Marika Sato Shigeaki Watanabe Hitomi Watanabe Gen Kondoh Tetsuya Kadonosono Shinae Kizaka-Kondoh Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases Communications Biology |
| title | Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases |
| title_full | Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases |
| title_fullStr | Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases |
| title_full_unstemmed | Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases |
| title_short | Tissue-resident macrophages are major tumor-associated macrophage resources, contributing to early TNBC development, recurrence, and metastases |
| title_sort | tissue resident macrophages are major tumor associated macrophage resources contributing to early tnbc development recurrence and metastases |
| url | https://doi.org/10.1038/s42003-023-04525-7 |
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