Deficiency of MGAT2 increases energy expenditure without high-fat feeding and protects genetically obese mice from excessive weight gain

Acyl CoA:monoacylglycerol acyltransferase 2 (MGAT2) is thought to be crucial for dietary fat absorption. Indeed, mice lacking the enzyme (Mogat2−/−) are resistant to obesity and other metabolic disorders induced by high-fat feeding. However, these mice absorb normal quantities of fat. To explore whe...

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Autors principals: David W. Nelson, Yu Gao, Nicole M. Spencer, Taylor Banh, Chi-Liang Eric Yen
Format: Article
Idioma:English
Publicat: Elsevier 2011-09-01
Col·lecció:Journal of Lipid Research
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Accés en línia:http://www.sciencedirect.com/science/article/pii/S0022227520408636

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