Deficiency of MGAT2 increases energy expenditure without high-fat feeding and protects genetically obese mice from excessive weight gain
Acyl CoA:monoacylglycerol acyltransferase 2 (MGAT2) is thought to be crucial for dietary fat absorption. Indeed, mice lacking the enzyme (Mogat2−/−) are resistant to obesity and other metabolic disorders induced by high-fat feeding. However, these mice absorb normal quantities of fat. To explore whe...
| Huvudupphovsmän: | , , , , |
|---|---|
| Materialtyp: | Artikel |
| Språk: | English |
| Publicerad: |
Elsevier
2011-09-01
|
| Serie: | Journal of Lipid Research |
| Ämnen: | |
| Länkar: | http://www.sciencedirect.com/science/article/pii/S0022227520408636 |