A connexin30 mutation rescues hearing and reveals roles for gap junctions in cochlear amplification and micromechanics
A point mutation in the gap-junction protein connexin 30 stops early onset age-related hearing loss. Here, the authors show that gap junctions contribute to cochlear micromechanics and that cochlear amplification is likely controlled by extracellular potentials in vicinity of the cochlear sensory ce...
| Main Authors: | , , , , |
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| 格式: | Article |
| 語言: | English |
| 出版: |
Nature Portfolio
2017-02-01
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| 叢編: | Nature Communications |
| 在線閱讀: | https://doi.org/10.1038/ncomms14530 |